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Bony disruption
Compound fractures
Simple fractures
Direct violence
Indirect violence
Pathological fracture
Osteoporosis
Secondary metastases
Fracture type
Depends on
Anatomic location Region (diaphysis , metaphysis, epiphysis,
intra/extra articular) Fracture lines (transverse, oblique, spiral) Condition of bone (comminuting, pathologic, incomplete, segmental)
Bone loss
Butterfly fragment Stress fracture
Diagnosis
1. Anamnesis
Mechanism of injury, history
A fracture is suspected from history and clinical examination, and confirmed by radiography
2.
Physical examination
Look
Asymmetry of contour Comparing one side with the other Displaced, angled Local bruising, swelling, laceration Asymmetry of posture Femoral neck fracture with external rotation Angulations, shortening
Feel
Crepitus, tenderness
Movement
false moving/ pseudoarthrosis
Neurovascular disturbance
Neurologic
Sensor and motor fx distal to fracture site
Vascular
Pulse palpation Capillary refill
Compartment syndrome
Pain
Pale
Parestesia Paralysis
Pulseless
3. Radiological examination
Two projection
Standard projections AP and lateral
Two articulation
Above and below , dislocation?
Two extremity
Comparison, especially in child
Two times
Hair line, callus formation
FRACTURE complications
Delayed union
Non union
Atrophic nonunion
Hypertrophic nonunion
Diabetic nonunion
Diabetic nonunion
Pseudoarthrosis nonunion
Mal union
Colles malunion
Debridement / guttering
Joint contracture, stiffness Limb shortening Compartment syndrome NVD Avascular necrosis Heterotrophic ossification
Management
Closed reduction
Splinting Casting Traction technique
Open reduction
Splintage
Allows sliding between implant and bone
Bridging
To bridge an area of comminution
Kirschner wire
Kirschner wire
Plate and
screw
External
fixator
Rehabilitation
Extremely important Part of fracture management Regain optimal function asap Arrangements
Restoring ROM Stretching Strengthening
Lower extremity
Cane Crutch walker
Fracture healing
Inflammation stage
Bleeding at fracture site Source of hemopoetic cells
Proliferation
Fibrovascular tissue developed Osteoblast, fibroblast proliferate
Repair stage
Primary callus or bridging callus occur 2wks Fibrocartilage developed Soft callus to hard callus (woven bone) by ossification Medullary callus can supplements the bridging callus
Remodeling stage
From midpoint repair until the fractures heal
clinically (up 7 yrs) Woven bone replaced by lamellar bone Bone assume its normal configuration
If there is anomaly in biological bone formation in healing process, there will be disturbance in the union of fractures, ex delayed or nonunion.
Thank you