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Definition
The acute abdomen may be defined generally as
an any sudden ,spontaneous intraabdominal
process causing severe abdominal pain and often
requiring surgical intervention.
It is a condition that requires a fairly immediate
judgement or decision as to management.
Decision requires
1.Patient ‘s history
2.Abdominal Examination /Physical finding
3.Laboratory data
4.Imaging test
CLINICAL DIAGNOSIS
1.Patient ‘s history
History-taking in the presence of the acute abdomen, which is
almost synonymous with “acute abdominal pain “
Abdominal pain that persists for 6 hours or more with severe intensity
increases the likelihood that surgical operation will be required.
If the pain ebbs after a few hours, however, the probability of surgical
disease decreases, but not to zero.
All the organs contained in the abdomen, pelvis and retroperitoneum can
be the cause of abdominal pain.
However, one must not forget that acute abdominal pain can also be
caused by some acute medical problems.
Abdominal pain is mediated through both the
1.Autonomic nervous systems.
2.Somatic sensory nervous systems.
Sign of PERITONITIS
This pain is often associated with the classic “peritoneal”
signs of rigidity: spasm of overlying muscles and rebound
tenderness from inflammation of the parietal peritoneum.
Nine sites of abdominal pain.
ABDOMINAL PAIN
Persist for 6 hours or more with severe intensity
increase- surgical operation
Ebbs after few hours the probability of surgical
diseases decreases, but not to zero
A.LOCATION OF PAIN
VISCERAL PAIN
(distension,inflamation,ischemiadull,poorly
midabdomen)
PARIETAL PAIN
( sharper and better localized, 4 kwadran )
REFERRED PAIN ( biliary diseases right shoulder)
SHIFTING PAIN
( acute appendicitis ,perforasi PUD—>epigastric –>right
kwd )
C.CHARACTER OF PAIN
STEADY PAIN
COLICKY PAIN
OTHER SYMPTOMS ASSOCATED WITH ABDOMINAL PAIN
A.VOMITING
CLEAR VOMITUS
BILE STAINED VOMITUS
FECULENT VOMITUS
B.CONSTIPATION
PARALYTIC ILEUS
MECHANICAL BOWEL OSTRUCTION
C.DIARRHEA
WATERY DIARRHEA
BLOOD STAINED DIARRHEA
D.OTHER SPESIFIC SYMPTOMS
JAUNDICE
HEMATEMESIS
HEMATOCHEZIA
MELENA
HEMATURIA
A.GYNECOLOGIC HISTORY
MENSTRUAL HISTORY - ECTOPIC PREGNANCY
VAGINAL DISCHARGED PID
B.DRUG HISTORY
CORTICOSTEROID
ANTI COAGULANT
NSAID
CLINICAL DIAGNOSIS
A.INSPECTION
B.AUSCULTATION
C.PERCUSSION
D.PALPATION
E. RECTAL EXAMINATION
F.EXAMINATION INGUINAL AND FEMORAL RINGS
G.COUGHING TO ELICIT PAIN
Specific signs in abdominal examination
Sign Description Pathology
3.LABORATORY INVESTIGATION
A.BLOOD STUDIES
B.URINE TEST
C.STOOL TEST
4.IMAGING STUDIES
A.PLAIN CHEST X RAY
B.PLAIN ABDOMINAL X RAY STUDIES
C.ANGIOGRAPHY
D.GASTROINTESTINAL CONTRAST X RAY STUDIES
E.ULTRASONOGRAPHY
F.CT SCAN
Acute Appendicitis
Etiology:
Obstruction of the lumen appendix followed by infection may
be from :
Catarrhal appendicitis.
lymphoid hyperplasia of the submucosal follicles 60% of
patients in children
-Gastro enteritis
-Virus
-Acute respiratory infection
-Mononucleosis
Obstructive appendicitis
-Fecalith 35% observed in adults.
-Foreign body / parasites (4%)
-Tumors (1%)
Etiology and Pathophysiology
Wangensteen proposed
1. Closed loop obstruction
bacterial overgrowth
2. Increase in luminal pressure.
Distention of the appendix
epigastric or periumbilical pain visceral pain
The overlying parietal peritoneum becomes irritated
classic migrating abdominal pain parietal pain and the
pain now localizes to the right lower quadrant (RLQ)
SHIFTING PAIN
3. Necrosis perforation occur about 48 hours .
If the body successfully walls off the perforation Appendiceal
Mass
If the perforation is not successfully walled off Diffuse
peritonitis will develop.
Differential diagnosis of acute appendicitis
Surgical Urological
• Acute Intestinal obstruction • Right ureteric colic
Blumberg sign
Rovsing sign
Obturator sign
Psoas sign
IVESTIGATIVE STUDIES
LABORATORY INVESTIGATION
-.BLOOD STUDIES
IMAGING STUDIES
A.ULTRASONOGRAPHY
B.CT SCAN
Complications
• PerforationGeneral Secondary Peritonitis
• Appendiceal Mass
• Appendiceal Abscess
• Pylephlebitis is suppurative thrombophlebitis of the
portal venous system
-chills
-high fever
-low grade jaundice
-hepatic abscess
TREATMENT
Resuscitated
Preoperative prophylactic antibiotics
Antibiotic treatment may be stopped.
-Becomes afebrile
-WBC count normalizes,
Appendiceal Mass
Nonoperative management
Periappendiceal infection becomes walled off by omentum and ajacent
viscera.
Initially treated with intravenous broad-spectrum antibiotic
- Perforation
- Infected (Inflammation )
Peritonitis are classified as :
1. Primary peritonitis
2. Secondary peritonitis
3. Tertiary peritonitis
1. Generalized peritonitis
2. Localized peritonitis
Primary peritonitis
Peritonitis occurring in the absence of an obvious source of
contamination , no pathologic process in a visceral organ
Tertiary peritonitis
- Anastomotic leakage
-Abscess with or without fistulization.
Clinical:
4. Hypovolemia Hypotensive
5.Auscultation
-Paralytic Ileus
Female patients
Vaginal and bimanual examination Pelvic inflammatory
disease
IVESTIGATIVE STUDIES
LABORATORY INVESTIGATION
Blood test
Liver function tests if clinically indicated
Serum electrolytes
Renal function
Amylase and lipase if pancreatitis is suspected
Urinalysis
Aerobic and anaerobic blood cultures
IMAGING STUDIES
Chest x-ray
Complications
• Hypovolaemia shock
• Electrolyte disturbances
• Peritoneal abscess
Vital Sign
Hypovolumic shock
Tachicardia
Hypotension
RR ↑
Fever
Oliguria
Altered mental status
Pathophysiology
Dependent upon :
1. Degree of obstruction
2. Duration of obstruction
3. Presence and severity of ischaemia
Result in :
1. Hypovolumic shock oliguria, hypotension,hemoconcentration
2. Electrolyte depletion
3. Bacterial overgrowth Rapid colonisation
-Maximal by 24 hrs after obstruction
-Bacterial translocation to node and portal system
4. Bowel distension Abdominal compatment syndrom
5. LBO
Ileocaecal valve plays prominent role in pathophysiology of LBO.
If competent valve = Closed loop obstruction
In 10 – 20 % of individual ICV incompetent
Caecal around 10 – 12 cm the risk of perforation
Abdominal Examination
Patient Supine position with the legs flexed at the hip
On Auscultation
Metallic sound
Borborygmi
On Palpation
Inguinal ,Femoral , Umbilical ,Incisional Hernias
Palpable mass
On Percuss
Dull Fluid or Mass
Tympanic Air (Intraluminal or not )
Peritoeal irritation
Overt
• Signs of upper GI sources of blood
-Hemetemesis
-Coffee-ground vomitus
• Blood per rectum occurs with any GI source
-Hematochezia
-Melena
Occult
-not clinically evident
FOBT
Iron deficiency anemia
Causes of Upper GI Bleed (UGIB)
Peptic Ulcer Disease (60% )
Erosive Gastritis/Esophagitis (15%)
Esophageal and Gastric Varices (6%)
Others
Stress ulcer
Arteriovenous malformation
Malignancy
4.PPI
1.HEMODINAMIC STABLE
Endoscopy diagnostic and therapeutic
Colonoscopy diagnostic and therapeutic
2.HEMODINAMIC UNSTABLE
SURGERY