Documenti di Didattica
Documenti di Professioni
Documenti di Cultura
By:
Krisnawati Ponggalunggu
C111 11 264
Supervisor :
Dr. dr. Khalid Saleh, Sp.PD-KKV, FINASIM
PATIENTS IDENTITY
Name
: Mr. B
Gender
: Male
Age
: 55 years old
Occupation
: Police
Registration no. : 740809
Date of Admission : 11/01/2016
History Taking
Chief Complaint:
Chest pain
Guided Anamnesis:
Occured about 9 hours before admitted to the hospital. The chest
pain was felt suddenly during rest. The pain was felt like
compressed pain and radiated to the the back. The duration of the
chest pain is about 30 minutes. The pain was also associated
with cold sweat. The history of chest pain before was denied.
Short of breathness during laying down was denied, no history of
coughing, fever, nausea and vomiting or epigastric pain. History
of short of breathness was also denied.
Defecation and urination were normal.
Risk Factors
Non-Modified Risk Factor :
Gender Male
Age > 45 years old
Modified Risk Factor :
Smoking (+) 1 pack a day
Clinical Examination
GENERAL STATE
Moderate illness/normo weight/conscious
VITAL SIGN
- Blood pressure : 110/70 mmHg
- Pulse: 86 times/min
- Breathing : 20 times/min
- Temperature : 36,6C (Axilla)
Head Examination
Inspection
: symmetric R=L
Palpation
: mass (-), tenderness (-), VF R=L
Percussion
: sonor R=L
lung-hepar border=right ICS IV
Right back lung border = right CV th VIII
Cardiac Examination
Inspection
: heart apex was not visible
Palpation
: heart apex was palpable
Percussion
:
Upper heart border :ICS II Linea parasternalis sinistra
Heart left border : ICS V Linea aksilaris anterior sinistra
Heart right border : ICS IV Linea parasternalis dextra
Auscultation
: Regular of I/II heart sound,
murmur (-)
Abdominal Examination
Inspection
: flat abdomen and following breath
movement
Auscultation
: peristaltic sound (+), normal
Palpation
: liver and spleen unpalpable
Percussion
: tympani (+), ascites (-)
Extremities
- Oedema
ELECTROCARDIOGRAM
Rhythm
Heart Rate
Regularity
P wave
PR interval
Axis
: sinus rhytm
: 75 bpm
: reguler
: 0.08 sec
: 0.24sec
: Normoaxis
INTERPRETATION
Rhythm
: sinus rhytm
Heart Rate : 75 bpm
Regularity : reguler
P wave
: 0.08 sec
PR interval : 0.24sec
Axis
: Normoaxis
Duration of QRS : 0.08 sec
ST segment : elevation in II,III, aVF, v7, v8, v2R-v3R; depression
in V2-V6
T wave
: normal T wave
Conclusion :
STEMI inferoposterior
RV Infarction
AV Block 1st degree
LABORATORY FINDINGS
TEST
RESULT
WBC
15,1 x 103
RBC
NORMAL
NORMAL
TEST
RESULT
Troponin I
>10,0
<0,01
4,09 x 106
CK
1720
<190
HGB
13,1
14 18
CKMB
69,5
<25
HCT
38
37 48
Natrium
137
136 145
PLT
205 x 103
Kalium
3,6
3,5 - 5,1
PT
11
10 14
Klorida
107
97 111
APTT
26,3
22,0 - 30,0
Asam Urat
4,3
3,4-7,0
INR
1,06
1,00
200
GDs
108
<110
Kolesterol HDL 54
>55
SGOT
26
<38
<130
SGPT
35
<41
Trigliserida
200
Ureum
39
10-50
Kreatinin
0,89
0,5-1,2
VALUE
110
VALUE
CHEST X-RAY
Conclusion:
Cardiomegaly
with signs of
pulmonary
congestive
WORKING DIAGNOSIS
ST Elevation Myocardial Infarction
(STEMI) Inferoposterior onset
<12hours
Right Ventricle Infarction
THERAPY
O2 2-4 liters/minute/NC
Antiplatelet
Simvastatin 20 mg 0-0-1
DISCUSSION :
DEFINITION
Myocardial infarction is myocardial
tissue damage caused by imbalance
between myocardial oxygen supply and
myocardial oxygen consumption.
Acute coronary syndromes include :
ST-elevation MI (STEMI)
Non ST-elevation MI ( NSTEMI)
Unstable Angina
RISK FACTORS
Modifiable
Pathophysiology
ATHEROSCLEROSIS OF CORONARY
ARTERY
SYMPTOMS
Typical
DIAGNOSIS
History taking
WHO Diagnostic
Criteria
Clinical history of ischaemic type
chest pain lasting >20 minutes
Changes in serial ECG tracings
Rise and fall of serum cardiac
biomarkers such as creatinine
kinase-MB fraction and troponin
http://en.wikipedia.org/wiki/Myocardial_infarction
Diagnosis
ECG CHANGES
Hyperacute
Phase
Complete
Evolution
Specific STElevation
T inverted
Q-Pathologic
Old Infarct
Q-Pathologic
ST segment
isoelectric
T normal or inverted
Treatment
Oxygen
Nitrate
Anti platelet agent
Anti koagulan
Morphine / pethidine
Trombolitic
-blocker
ACE inhibitors
Lipid lowering agent
Surgical
revascularization
PTCA (percutaneous
transluminal coronary
angioplasty)
CABG (coronary artery
bypass grafting)
Prognosis
KILLIP CLASSIFICATION
Clas
Description
s
I
no clinical signs of heart
failure
II
rales or crackles in the lungs,
an S3, and elevated jugular
venous pressure
III
acute pulmonary edema
IV
cardiogenic shock or
hypotension (systolic BP < 90
mmHg), and evidence of
peripheral vasoconstriction
http://en.wikipedia.org/wiki/Killip_class
Mortality Rate
(%)
6
17
30 - 40
60 80
Thank You