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BIOLOGIC
CRISIS
OBJECTIVES
At the end of 3H lecture, the students will
be able to:
1. Demonstrate standard assessment
methods & principles & techniques of
physical examination of the critically ill
patients,
2. analyze and interpret the different noninvasive and invasive diagnostic & lab
tests results of clients with multi-organ
problems,
ACUTE BIOLOGIC
CRISIS
Any conditions that may result
to patient mortality if left
unattended in a brief period of
time.
Standard Assessment
and
Primary Assessment
Secondary Assessment
Primary Assessment
Primary Assessment
A-airway (patency)
B- breathing (effectiveness)
C- circulation (both peripheral & organ specific)
For trauma patients, the cervical spine area must be
evaluated
If the ABC status is satisfactory, the SECONDARY
ASSESSMENT is performed.
Secondary Assessment
Identifies
A. NEUROLOGIC ASSESSMENT
Determine clients:
1.
Level of consciousness (LOC)
2.
Orientation to person, place, time & event
3.
Glasgow coma scale (GCS) score
4.
Pupillary size, equality & reaction to light and
accommodation (PERRLA)
5.
Motor movement & strength of hand grips &
pedal pushes
*in children, brief neurologic assessment can be
determined using AVPU mnemonic
CHILD NEUROLOGIC
ASSESSMENT
GCS
B. HISTORY
clients chief complaint,
duration of the problem,
mechanism of injury,
past pertinent medical history,
current medications,
use of alcohol,
known allergies and immunizations.
For women, OB history is obtained.
SCENARIO:
D. PAIN
PQRST mnemonic
P provokes
Q- quality (type of pain)
R- Region/Radiation (Location of pain)
S- Severity (pain scale of 1-10, visual analogs)
T- Timing (how long?)
*pain rate of 7 & above is severe and is considered high
risk.
SITUATION:
C. GENERAL OVERVIEW
note clients over-all health condition, skin color,
gait, posture, unusual skin markings or body
odors, mood and affect.
Measurement of vital signs and O2 saturation
are obtained as well.
E. HEAD-TO-TOE FOCUSED
ASSESSMENT
Examine
Aorta
Pulmonary
arteries
Pulmonary
veins
Pulmonary
veins
ELECTROCARDIOGRAM
Essential
PRE-PROCEDURE CARE
1. Explain that the test helps evaluate the hearts
function by recording its electrical activity.
2. Remove all metal objects in the body, prevent body
from touching any metal.
3. Steps for ECG monitoring:
A. attach the electrodes to the clients skin
B. connect the electrodes to the monitor by a cable
C. Adjust the monitor/leads to obtain a readable ECG
3. During the procedure, advise the client to stand still,
breath normally, and refrain from talking.
4. Record the clients age, height, weight and note any
cardiac medications being taken.
12 LEAD ELECTROCARDIOGRAM
1.
2.
6 Chest Leads
4 limb or peripheral
leads (one of these
is "neutral")
UNIPOLAR LEADS
The "AV" stands for "Augmented Vector".
The last letter refers to position, which are as
follows:
Label
Meaning of label
Position of lead
on body
AVr
Right wrist
AVL
Left wrist
AVf
Left foot
BIPOLAR LEADS
Lead
aV
r
L
V
a
aVf
POST-PROCEDURE CARE
1. After the procedure, disconnect the
equipment.
2. If using a conductive gel, wipe the gel
from the clients skin.
3. If using conductive stickers, remove
unless serial ECG readings are to be
done.
SINUS RYTHM
SUMMARY
P - atrial depolarization
T - ventricular repolarization
U - follows T-wave (hypokalemia)
HOLTER MONITORING
Ambulatory electrocardiography
A continuous ECG tracing is recorded
continuously for a day or longer on an outpatient
basis.
Detects dysrhythmias that may not appear on a
routine ECG but occur when the client is
ambulating at home or work.
Useful in evaluating effectiveness of
antidysrhythmic or pacemaker therapy.
PRE-PROCEDURE CARE
1.
2.
CARDIAC RHYTHMS
CARDIAC RHYTHMS
Sinus
Bradycardia
Sinus Tachycardia
Cardiac Dysrhythmias
U-wave
Atrial fibrillation
Atrial tachycardia
Ventricular tachycardia
Ventricular fibrillation
THE U WAVE
Another wave after the PQRST complex.It is not very
common and is easy to overlook.
Found in severe hypokalemia
ATRIAL FIBRILLATION
an irregular and often rapid heart rate that
commonly causes poor blood flow to the body
the heart's two upper chambers (the atria) beat
chaotically and irregularly out of coordination
with the two lower chambers (the ventricles) of
the heart
symptoms include heart palpitations, shortness
of breath and weakness
usually isn't life-threatening, it is a serious
medical condition that sometimes requires
emergency treatment
absence of P-wave
ATRIAL FLUTTER
Your heart's upper chambers (atria) beat too
quickly.
caused by problems in your heart's electrical
system.
200-350 beats per minute
a saw-toothed waveform
VENTRICULAR TACHYCARDIA
A rapid heartbeat that starts in the ventricles.
Pulse rate of more than 100 beats per minute,
with at least three irregular heartbeats in a row.
The condition can develop as an early or late
complication of aheart attack. It may also occur
in patients with:
Cardiomyopathy
Heart
failure
Heart surgery
Myocarditis
Valvular
heart disease
VENTRICULAR FIBRILLATION
A severely abnormal heart rhythm that can be
life-threatening.
Interruption of the heartbeat for only a few
seconds can lead to fainting (syncope) or cardiac
arrest.
Fibrillation is an uncontrolled twitching or
quivering of muscle fibers (fibrils).
Blood is notpumped from the heart. Sudden
cardiac death results.
VENTRICULAR TACHYCARDIA
VENTRICULAR FIBRILLATION
EXERCISE:
ACTIVITY
Draw the ECG tracing of the following and write a
short description about it
1.
2.
3.
4.
HEMODYNAMIC STATUS
Assessed
1.
2.
3.
4.
in four parameters:
Proximal
Distal lumen
Small third lumen
Fourth lumen
Fifth port
Lumen
Location
Uses
Proximal lumen
Right
atrium
Distal lumen
inflation/deflation of balloon
computer
thermistor port, CO
infusion of fluids, O2 sat
2.
3.
Watch this
SWAN GANZ CATHETER INSERTION
Decrease in CVP
Increase in CVP
1. decrease in circulating
volume which may result
from fluid imbalance,
2. hemorrhage or severe
vasodilation and
3. pooling of blood in the
extremities with limited
venous return
MEASURING CVP
CVP READING
Increased PCWP
insufficient volume
Insufficient pressure in
the left ventricle
pulmonary congestion
hypovolemic shock
Increased CO
Hypoxia
Shock
Exercise
Heat stroke
IVF intake
Dysrhythmias
Septic shock
Arterial hemorrhage
Myocardial ischemia
ABG
ANALYSIS OF ABG
1. Classify the pH
(Normal 7.34-7.45)
Acidemia: 7.35
Alkalemia: 7.45
2. Assess PaCO2
(Normal 35-45mmHg)
Respiratory acidosis: 45 mmHg
Respiratory Alkalosis: 35 mmHg
3. Assess HCO3
(Normal 22-26 meq/L)
Metabolic acidosis: 22 meq/L
Metabolic alkalosis: 26 meq/L
4. Determine presence of compensation
Compensation present: paCO2 & HCO3 are abnormal
Compensation absent: only one component is abnormal
5. Identify primary disorder- where the pH is leaning
towards is probably the primary cause
R
O
espiratory
opposite
M etabolic
E equal
ABG EXERCISE
1.
pH
7.20
PaCO2 50 mmHg
HCO3 18 meq/L
3.
pH
7.6
PaCO2 45 mmHg
HCO3 30 meq/L
2.
pH
7.13
PaCO2 49 mmHg
HCO3 24 meq/L
4.
pH
7.49
PaCO2 32 mmHg
HCO3 30 meq/L
End of Part 1
Interlude