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HOST: Estrogen production and the presence of low vaginal PH Abnormal activity of Hh signaling pathway (Hh)
AGENT: Idiopathic/unknown Malfunctioning of the FOX L2 gene (Fork head box protein) ENVIRONMENT:
Increasing age
Stress and stimulation within the pelvic cavity stimulates the fibroma
Dysplasia
Inflammation process
Natural killer cells, killer T cells, dendritic cells and macrophages move towards the tumor site Production of Chemokines CXCL10, CXCL9 and CXCL 11
Inhibit formation of new blood vessels Induces growth of death Increase tumor cell ovarian fibroma Increase antibody production
Circulation Ingestion of debris by dendritic cells and move towards draining lymphnodes
Tumor specific dendritic cells trigger differentiation of Th1 cells Development of CD8 + T cells
CD4 and CD8 + T cells & cytolytic T cells destroy the tumor cells remaining at the site
hyperestrogenism
Vaginal spotting
Abdominal discomfort
ascites
http://www.scribd.com/doc/17600979/Pa-Tho-Physiology-of-Uterine-Myoma http://nursing-resource.com/uterine-febroids/ http://uk.ask.com/beauty/Pathophysiology-of-Myoma-Uteri http://www.scribd.com/doc/50267879/33880230-Uterine-Myoma http://www.scribd.com/doc/38186951/Myoma-Case-Study http://www.scribd.com/doc/48493187/Uterine-MYOMA http://www.scribd.com/doc/50267879/4/IV-NARRATIVE-PATHOPHYSIOLOGY http://www.scribd.com/doc/6333556/Diagram-Myoma-i http://www.scribd.com/doc/6333558/Diagram-Myoma-II http://www.scribd.com/doc/6333561/Diagram-Myoma-III