PNF Techniques help develop muscular strength and endurance, joint stability, mobility, neuromuscular control and coordinationall

of which are aimed at improving the overall functional ability of patients. Developed in the 1940s, PNF Techniques are the result of work by Kabat, Knott and Voss.They combined their analysis of functional movement with theories from motor development, motor control, motor learning and neurophysiology. PNF History: In the early to mid 1900s physiologist Charles Sherrington popularized a model for how the neuromuscular system operates. Irradiation is when maximal contraction of a muscle recruits the help of additional muscle flexibility. Based on that, Herman Kabat, a neurophysiologist, began in 1946 to look for natural patterns of movement for rehabilitating the muscles of polio patients. He knew of the myostatic stretch reflex which causes a muscle to contract when lengthened too quickly, and of the inverse stretch reflex, which causes a muscle to relax when its tendon is pulled with too much force. He believed combinations of movement would be better than the traditional moving of one joint at a time. To find specific techniques, he started an institute in Washington, DC and by 1951 had two offices in California as well. His assistants Margaret Knott and Dorothy Voss in California applied PNF to all types of therapeutic exercise and began presenting the techniques in workshops in 1952. During the 1960s, the physical therapy departments of several universities began offering courses in PNF and by the late 1970s PNF stretching techniques began to be used by athletes and other healthy people for more flexibility and range of motion. Terms about muscle contraction are commonly used when discussing PNF.
PNF - Techniques

Basic PNF Techniques: Revarsal of Antagonists: A group of techniques that allow for agonist contraction followed by antagonist contraction without pause or relaxation.

Dynamic Reversals (Slow Reversals): Utilizes isotonic contractions of first agonists, then antagonists performed against resisitance . Contraction of stronger pattern is selected first with progression to weaker pattern. The limb is moved through full range of motion. Indications Impaired strength and coordination between agonist and antagonist, limitations in range of motion, fatigue. Stabilizing Reversals: Utilizes alternating isotonic contractions of first agonists, then antagonists against resistance, allowing only very limited range of motion. Indications Impaired strength, stability and balance, coordination. Rhythmic Stabilization (RS): Utilizes alternating isometric contractions of first agonists, then antagonists against resistance; no motion is allowed . Indications Impaired strength and coordination, limitations in ROM; impaired stabilization control and balance.

Repeated Contractions, RC (Repeated stretch): Repeated isotonic contractions from the lengthened range, induced by quick stretches and enhanced by resistance; performed through the

range or part of range at a point of weakness. Technique is repeated (i.e.,three or four stretches) during one pattern or until contraction weakens. Indications Impaired strength, initiation of movement, fatigue and limitation in active ROM. Combination of Isotonics (Agonist Reversals, AR): Resisted concentric, contraction of agonist muscles moving through the range is followed by a stabilizing contraction (holding in the position) and then eccentric, lengthening contraction, moving slowing back to the start position; there is no relaxation between the types of contractions. Typically used in antigravity activities/assumption of postures (i.e., bridging, sit to stand transitions). Indications Weak postural muscles, inability to eccentrically control body weight during movement transitions, poor dynamic posture control. Rhythmic Initiation (RI): Voluntary relaxation followed by passive movements progressing to active assisted and active resisted movements to finally active movements. Verbal commands are used to set the speed and rhythem of the movements. Light tracking is used during the resistive phase to facilitate movement. Indications Inability to relax, hypertonicity (spasticity, rigidity); difficulty initiating movement; motor planning deficits (apraxia or dyspraxia); motor learning deficits; communication deficits (aphasia). Contract Relax (CR): It is one of PNF Techniques usually performed at a point of limited ROM in the agonist pattern. Strong, small range isotonic contraction of the restricting muscles (antagonists) with emphasis on the rotators is followed by an isometric hold. The contraction is held for 5-8 seconds and is then followed by voluntary relaxation and movement into the new range of the agonist pattern. Movement can be passive but active contraction is preffered.

Contract-relax-active-contraction (CRAC): Active contraction into the newly gained range serves to maintain the inhibitory effects through reciprocal inhibition. Indications Limitation in ROM.

Hold Relax (HR): It is one of PNF Techniques usually performed at a position of comfort and below a level that causes pain. Strong isometric contraction of the restricting muscles (antagonists) is resisted, followed by voluntary relaxation, and passive movement into the newly gained range of the agonist pattern.

Hold-relax-active-contraction (HRAC): Similar to HR except movement into the newly gained range of the agonist pattern is active, not passive. Active contraction serves to maintain the inhibitory effects through reciprocal inhibitions. Indications Limitation in PROM with pain.

pnf techniques Replication (Hold Relax Active Motion, HRA): The patient is positioned in the shortened range/ end position of a movement and is asked to hold. The isometric contraction is resisted followed by voluntary relaxation and passive movement into the lengthened range. The patient is then instructed to move back into the end position; stretch and resistance are applied to facilitate the

isotonic contraction. For each repitition, increasing ROM is desired. Indications Marked weakness, inability to sustain a contraction in the sustained range. Resisted Progression (RP): Stretch, approximation and tracking resistance is applied manually to facilitate pelvic motion and progression during locomotion; the level of resistance is light so as to not disrupt the patient's momentum, coordination and velocity. RP can also be applied using elastic band resistance. Indications Impaired timing and control of lower trunk/pelvic segments during locomotion, impaired endurance. Rhythmic Rotation (RRo): Relaxation is achieved with slow, repeated rotation of a limb at a point where limitation is noticed. As muscles relax the limb is slowly and gently moved into the range. As a new tension is felt, RRo is repeated. The patient can use active movements (voluntary efforts) for RRo or the therapist can perform RRo passively. Voluntary relaxation when possible is important. Indications Relaxation of excess tension in the muscles (hypertonia) combined with PROM of the range-limiting muscles. To that end, PNF Techniques have broad applications in treating people with neurologic and musculoskeletal conditions, most frequently in rehabilitating the knee, shoulder, hip and ankle

. Bell's Palsy as most common anomaly of 7th cranial nerve Bell's palsy/Facial palsy is a form of facial paralysis resulting from damage to the 7th cranial nerve. Bell's palsy gets its name from the 19th century Scottish surgeon, Sir Charles Bell, who was the first to describe the condition ("Palsy" is an archaic (old) word which means paralysis). Bell's Palsy is the most common cause of facial paralysis in the world. The condition afflicts around 40,000 Americans each year. The incidence rate in the US is around 20 - 30 cases per 100,000 of the population. Worldwide, Bell's Palsy afflicts approximately 10 - 30 people per 100,000 of the population.

Anatomy of Facial nerve or 7th cranial nerve The 7th cranial nerve is mixed nerve containing both sensory and motor components. It emerges from the brainstem between the pons and the medulla, and controls the muscles of facial

expression, and functions in the conveyance of taste sensations from the anterior two-thirds of the tongue and oral cavity. 7th cranial nerve also supplies preganglionic parasympathetic fibers to several head and neck ganglia. The motor part of the 7th cranial nerve arises from the facial nerve nucleus in the pons while the sensory part of the 7th cranial nerve arises from the nervus intermedius. The motor part and sensory part of the 7th cranial nerve enters the petrous temporal bone into the internal auditory meatus (intimately close to the inner ear) then runs a tortuous course (including two tight turns) through the facial canal, emerges from the stylomastoid foramen and passes through the parotid gland, where it divides into five major branches. Though it passes through the parotid gland, it does not innervate the gland (This is the responsibility of cranial nerve IX, the glossopharyngeal nerve). The 7th cranial nerve forms the geniculate ganglion prior to entering the facial canal. Branches of 7th cranial nerve

Greater petrosal nerve - provides parasympathetic innervation to lacrimal gland, sphenoid sinus, frontal sinus, maxillary sinus, ethmoid sinus, nasal cavity, as well as special sensory taste fibers to the palate via the Vidian nerve. Nerve to stapedius - provides motor innervation for stapedius muscle in middle ear. Chorda tympani - provides parasympathetic innervation to submandibular gland and sublingual gland and special sensory taste fibers for the anterior 2/3 of the tongue.

Outside skull (distal to stylomastoid foramen)

Posterior auricular nerve - controls movements of some of the scalp muscles around the ear. Branch to Posterior belly of Digastric and Stylohyoid muscle.

Five major facial branches (in parotid gland) - from top to bottom:

Temporal (frontal) branch of the 7th cranial nerve. Zygomatic branch of the 7th cranial nerve. Buccal branch of the 7th cranial nerve. Marginal mandibular branch of the 7th cranial nerve. Cervical branch of the 7th cranial nerve.

Function Efferent Its main function is motor control of most of the muscles of facial expression. It also innervates the posterior belly of the digastric muscle, the stylohyoid muscle, and the stapedius muscle of the middle ear. All of these muscles are striated muscles of branchiomeric origin developing from the 2nd pharyngeal arch.

The 7th cranial nerve also supplies parasympathetic fibers to the submandibular gland and sublingual glands via chorda tympani. Parasympathetic innervation serves to increase the flow of saliva from these glands. It also supplies parasympathetic innervation to the nasal mucosa and the lacrimal gland via the pterygopalatine ganglion.The 7th cranial nerve also functions as the efferent limb of the corneal reflex and the blink reflex. Afferent In addition, it receives taste sensations from the anterior two-thirds of the tongue via the chorda tympani, taste sensation is sent to the gustatory portion of the solitary nucleus. General sensation from the anterior two-thirds of tongue are supplied by afferent fibers of the third division of the fifth cranial nerve. These sensory and taste (VII) fibers travel together as the lingual nerve briefly before the chorda tympani leaves the lingual Nerve to enter the middle temporal fossa via the petrotympanic fissure. It thus joins the rest of the 7th cranial nerve in the internal acoustic meatus before synapsing in the solitary nucleus. The cell bodies of the Chorda tympani reside in the geniculate ganglion, and these parasympathetic fibers synapse at the submandibular ganglion, attached To the lingual nerve. The 7th cranial nerve also supplies a small amount of afferent innervation to the oropharynx below the palatine tonsil. There is also a small amount of cutaneous sensation carried by the nervus intermedius from the skin in and around the auricle (earlobe). Location of Cell Bodies The cell bodies for the 7th cranial nerve are grouped in anatomical areas called nuclei or ganglia. The cell bodies for the afferent nerves are found in the geniculate ganglion for taste sensation. The cell bodies for muscular efferent nerves are found in the facial motor nucleus whereas the cell bodies for the parasympathetic efferent nerves are found in the superior salivatory nucleus. Testing the facial nerve

Voluntary facial movements, such as wrinkling the brow, showing teeth, frowning, closing the eyes tightly (inability to do so is called lagophthalmos) , pursing the lips and puffing out the cheeks, all test the facial nerve. There should be no noticeable asymmetry. In an UMN lesion, called central seven, only the lower part of the face on the contralateral side will be affected, due to the bilateral control to the upper facial muscles (frontalis and orbicularis oculi). Lower motor neuron lesions can result in a 7th cranial nerve palsy (Bell's palsy is the term used to describe the idiopathic form of facial nerve palsy), manifested as both upper and lower facial weakness on the same side of the lesion. Taste can be tested on the anterior 2/3 of the tongue. This can be tested with a swab dipped in a flavoured solution, or with electronic stimulation (similar to putting your tongue on a battery).

Corneal reflex. The afferent arc is mediated by the General Sensory afferents of the Trigeminal Nerve. The efferent arc occurs via the Facial Nerve. The reflex involves consensual blinking of both eyes in response to stimulation of one eye. This is due to the Facial Nerve's innervation of the muscles of facial expression, namely Orbicularis Oculi, responsible for blinking. Thus, the corneal reflex effectively tests the proper functioning of both Cranial Nerves V and VII.

Cause of Bell's Palsy The etiology of this form of facial nerve palsy is unknown and therefore exactly what causes this damage is also unknown.

Many scientists believe that a viral infection such the common cold sore virus, herpes simplex, causes the disorder. It is thought that the 7th Cranial nerve (VII), which supplies the face, swells and becomes inflamed in reaction to an infection, causing pressure within the Fallopian canal, the narrow 'tunnel' in the skull through which the facial nerve passes in a somewhat tortuous route. As the nerve swells, it compresses against the inside of the Fallopian canal and as a result, the nerve is unable to function. Bell's Palsy has also been associated with herpes, mumps, influenza or a flu-like illness, headaches, chronic middle ear infection, high blood pressure, diabetes, tumors, Lyme disease, tuberculosis, trauma such as skull fracture, facial injury and even in some instances, dental work, if the facial nerve is damaged or frozen during injection. During pregnancy, Bell's palsy is sometimes associated with pre-eclampsia, a condition that affects women at an advanced stage of pregnancy, noted by high blood pressure, swelling of the ankles and protein in the urine. Bell's palsy is also more rarely associated with serious neurological conditions such as Guillain-Barre syndrome, neurosarcoidosis and multiple sclerosis.

Bell's Palsy and Pregnancy Bell's palsy is more common in pregnant women than in women who are not pregnant. Developing Bell's Palsy during pregnancy does not automatically indicate the presence of a more serious underlying condition. High blood pressure, diabetes, influenza or flu-like illness, middle ear infection and dental work, have all been associated with Bell's Palsy. During pregnancy, Bell's palsy is sometimes associated with pre-eclampsia, a condition that affects women at an advanced stage of pregnancy, noted by high blood pressure, swelling of the ankles and protein in the urine. This possibility should be investigated by the physician. Pre-eclampsia is a serious condition and careful management is required should this be the case. The condition, Bell's Palsy, poses no risk to the unborn infant, however it should always be investigated to eliminate any underlying conditions.

Symptoms of Bell's Palsy

Sometimes you may have a cold shortly before the symptoms of Bell's palsy begin. Symptoms most often start suddenly, but may take 2 - 3 days to show up. They do not become more severe after that. Symptoms are almost always on one side only. They may range from mild to severe. The face will feel stiff or pulled to one side, and may look different. Other symptoms can include:

Difficulty eating and drinking; food falls out of one side of the mouth Drooling due to lack of control over the muscles of the face Drooping of the face, such as the eyelid or corner of the mouth Hard to close one eye Problems smiling, grimacing, or making facial expressions Twitching or weakness of the muscles in the face

Other symptoms that may occur:

Dry eye or mouth Headache Loss of sense of taste Sound that is louder in one ear (hyperacusis) Twitching in face

Bell's Palsy Treatment Often, no treatment is needed. Symptoms often begin to improve right away. However, it may take weeks or even months for the muscles to get stronger, and this may be frustrating. Your health care provider may give you lubricating eye drops or eye ointments to keep the surface of the eye moist if you cannot close it completely. You may need to wear an eye patch while you sleep. Sometimes medicines may be used, but it is not clear how much they help. If medicines are used, they should be started right away.

Corticosteroids may reduce swelling around the facial nerve Medications can fight the virus that may be causing Bell's palsy

Surgery to relieve pressure on the nerve (decompression surgery) is controversial and has not been shown to routinely benefit people with Bell's palsy. Physiotherapy for Facial Palsy facial nerve Palsy causes temporary partial paralysis of facial muscles. Without treatment, recovery occurs for nearly all patients within one year and many patients recover within one month. Many patients chose to use physiotherapy treatment to help progress their recovery. If you want to use physiotherapy, you can choose to try

bell palsy facial exercises

electrical stimulation Manual massage Kabat rehabilitation

bell palsy facial exercises Patients most often chose facial exercises as their main form of physiotherapy treatment for Bell's Palsy. Facial muscles atrophy very slowly, so you do not need to exercise to maintain muscles as you would for a broken arm or leg. Instead, you will use facial exercises to keep your brain trained in what electrical impulses are needed to control the different muscles in your face. This will help you transition back to using your facial muscles as you recover from Bell's Palsy and the paralysis goes away. Facial exercises for Bell's Palsy involve doing basic actions with the different muscle groups throughout your face. If you are seeing a doctor or physical therapist for your Bell's Palsy, he will be able to recommend specific exercises appropriate for your level of paralysis. If not, try thinking about the different muscles in your face, and exercising them from top to bottom. Furrow your brow, raise your eyebrows, slowly blink your eyes, and continue down to your chin. When performing these exercises, you should not exert yourself or worry much about whether you are successfully completing all of them. Instead perform the exercises with an ability that matches your natural recovery from Bell's Palsy. Remember that the exercises are intended to work your mind more than they are intended to work your muscles. Facial exercises can be preformed at home. You should go through the exercises three times each day. EMG feedback and/or specific mirror exercises will provide a sensory feedback to promote learning. When each muscle group is being assessed, the patient observes the action of these muscles in the mirror and instructed to perform small symmetrical specific movements on the sound side to identify the right response. Each patient presents with different functional disability, so there are no general list of exercises. As patient identifies the specific area of dysfunction, patient can begin to perform exercise to improve facial movements' guided by the affected side so isolated muscle response is preserved and coordination improved. Repetitions & frequency of exercises can be modified according to improvement status. Synkinesis: The site of synkinesis should be identified in order to teach the patient how to control abnormal movement pattern. The treatment of synkinesis depends on inhibition of the unwanted movements that occur during volitional & spontaneous movements; by perform facial movement slowly without triggering the abnormal movement. Stretching is recommended here to prevent muscles tightness. Electrical Stimulation Some doctors recommend using electrical stimulation and biofeedback as an additional method of physiotherapy for Bell's Palsy, but other doctors believe that it does little to help recovery.

Decide with your doctor or therapist whether electrical stimulation is right for you. Unlike facial exercises which you mostly perform at home, you will need to visit a therapist for electrical stimulation. Electrical stimulation uses small amounts of electricity to activate the muscles in your face. This causes your muscles to twitch as if electrical impulses from your brain activated them. During the stages of Bell's Palsy when your face is most paralyzed, this may be the only way for you to activate these facial muscles. Manual Massage Massage can be performed in conjunction with other treatment options. It can be done to improve perceptual awareness. Massage manipulations on the face include:

Effleurage Finger or thumb kneading Hacking Tapping Stroking

Kabat Rehabilitation Kabat rehabilitation is type of motor control rehabilitation technique based on proprioceptive neuromuscular facilitation (PNF). During Kabat, therapist facilitate the voluntary contraction of the impaired muscle by applying a global stretching then resistance to the entire muscular section and motivate action by verbal input and manual contact. When performing Kabat, 3 regional are considered: the upper (forehead and eyes), intermediate (nose), and lower (mouth). Prior to Kabat, ice stimulation has to perform to a specific muscular group, in order to increase its contractile power. Prognosis Most cases go away completely within a few weeks to months. If you did not lose all of your nerve function and symptoms began to improve within 3 weeks, you're more likely to regain all or most of the strength in your facial muscles. Sometimes, the following symptoms still may be present:

Long-term changes in taste Spasms of muscles or eyelids Weakness that remains in facial muscles

Complications Excess drying of the eye surface, leading to eye ulcers or infections. Tips For Coping With Bell's Palsy

The Eye The main concern is to ensure the eye is protected and is kept moistened. At night-time, it is probably best to use a soft, surgical, eye patch, taped to ensure the eye remains closed during sleep. A small piece of lightly adhesive surgical tape on the eyelid itself may also be required. It is recommended that an eye patch be worn to prevent corneal erosion or other eye damage. During the day, when an eye patch is perhaps an inconvenience, although not recommended, the eyelid of the affected eye can be manually closed and 'blinked' several times. This is best done by use of soft, clean, tissues to gently close the eyelid. Protection from dust and dirt can be achieved by wearing suitable glasses although there is the risk of wind-blown dust and dirt entering the eye. Eating and Drinking Eating and especially drinking, can be rather awkward with Bell's palsy. The only effective method of ensuring that the food / liquid remains in the mouth is to physically hold the affected corner of the mouth closed by holding both lips between thumb and forefinger on the affected side. It may not be particularly attractive or graceful but it is better than wearing what you were trying to eat or drink!