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ETIOLOGIES: SECONDARY:
PRIMARY: Systemic Lupus Erythematosus Hepatitis
Infection: Pyelonephritis Diabetes Mellitus Malaria
Glomerulopnephritis Allergic Responses Cyanotic Heart Disease
Sickle Cell Anemia Tuberculosis
Anaphylactoid Purpura Infected Vedntriculojugular shunts
Renal Vein Throimbosis Stings/Venoms
Drug Toxicity: TRIMETHADIONE
IgG Level Falls Endothelial lining and basement membranes damaged (Renal Glomeruli Damage) Steroids
Activates Renin-Angiotensin Fluid level accumulates in interstitial spaces and body cavities Albumin IV Transfusion
System Hypertension Weight Gain
EDEMA Abdomen Ascites
Antihypertensive Drugs
Adrenal Secretion of Aldosterone Eyes Periorbital Edema
Monitor BP Weigh Daily and dietary
Scrotum
Impaired skin integrity restrictions
related to the Increase RBC and Platelet
Vasoconstriction presence of edema as Excess fluid volume related to compromised renal
evidenced by perfusion as evidenced by decreased urine output
Lack of knowledge of the mother Clots Form and edema
reddened or taut skin
Increase absorption of Sodium and
about the disease entity or actual breaks in the
water in distal tubules skin Blood flow slows Acute pain related to presence LEGENDS:
of edema as evidence by Classical Signs
Knowledge deficit regarding condition, prognosis, complaints of pain, and wincing Physiology changes
treatment, self-care, and discharge needs related to Clotting Problem Arise on movement Clinical Manifestations
lack of exposure
Treatment or Nursing
Decrease blood flow to kidneys Interventions
Nursing Diagnoses
End Stage Renal Failure Dialysis