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  • Pharma 14

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    Maica Aquino
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    This document summarizes the pathophysiology of a myocardial infarction (MI). Key points include: an MI occurs when there is obstruction of blood flow to the heart muscle, usually due to a thrombus or embolism, leading to injury and death of myocardium. Diagnosis involves ECG changes and elevated cardiac enzyme levels. The renin-angiotensin-aldosterone system and other mechanisms aim to regulate blood pressure in response to an MI by influencing vascular tone, fluid volume, and other factors.

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    This document summarizes the pathophysiology of a myocardial infarction (MI). Key points include: an MI occurs when there is obstruction of blood flow to the heart muscle, usually due to a thrombus or embolism, leading to injury and death of myocardium. Diagnosis involves ECG changes and elevated cardiac enzyme levels. The renin-angiotensin-aldosterone system and other mechanisms aim to regulate blood pressure in response to an MI by influencing vascular tone, fluid volume, and other factors.

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    © All Rights Reserved
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    Segnala contenuti inappropriati
    12 visualizzazioni2 pagine

    Pharma 14

    Caricato da

    Maica Aquino
    This document summarizes the pathophysiology of a myocardial infarction (MI). Key points include: an MI occurs when there is obstruction of blood flow to the heart muscle, usually due to a thrombus or embolism, leading to injury and death of myocardium. Diagnosis involves ECG changes and elevated cardiac enzyme levels. The renin-angiotensin-aldosterone system and other mechanisms aim to regulate blood pressure in response to an MI by influencing vascular tone, fluid volume, and other factors.

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    MYOCARDIAL INFARCTION

     Levine’s sign
     OBSTRUCTION
     Irreversible

    Atheroma Thrombus Embolus Coagulation Cascade

    INJURY
    OBSTRUCTION
    Vasoconstriction
    No Blood Flow
    Formation of platelet plug
    Injury to tissue
    Intrinsic pathway extrinsic pathway
    Death of myocardium
    Liver prothrombin thrombin
    Non- functioning
    (scar) Fibrinogen

    Fibrin
    LAB TEST: Angina MI
    ECG ST- STEM- CLOT
    depression STEM
    CARDIAC
    ENZYMES (-) (+)
    - myoglobin
    - troponin
    - CKMB
    - LDH

    Nursing Priority: PAIN


    BLOOD PRESSURE 4. RAAS
     Resistance/ pressure exerted by the Hypoxia KIDNEY
    blood on the vascular smooth - secrete
    muscle. O2 supply Renin
     (blood to the wall) hormone
    Systemic blood
    BP= 120/80 mmHg flow (perfusion)

    BP = HR x SV x TPR x

    Cardiac output = HR x SV LIVER


    (angiotensino
    Stroke Volume- amount of blood
    gen
    ejected/BEAT Blood - produce A1
    flow back
    Total Peripheral Resistance- pressure
    to the
    exerted by smooth muscle against
    kidney A2
    the blood. (Wall to blood) A2 LUNGS
    receptor
    Angiotensin
    REGULATORS OF BP: Converting
    Enzyme
    1. Baroreceptors (aorta and carotid A.) vasoconstriction
    BP - A1 to A2
    BP
    (potent
    vasoconstrictor)

    Baroreceptors send signal to brain

    TPR ADRENAL
    HR CORTEX
    (A2 receptor)
    Blood volume - A2 to A3
    BP

    2. Vascular Autoregulation H2O Na+ Aldosterone


    BP > vasoconstriction > BP retention retention
    BP > vasodilation > BP High
    osmolarity
    3. Regulation of Body Fluid Volume (viscous)

    Body Fluid Volume ADH HYPOTHALAMUS


    produce

    BP

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