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CARE PLAN
ON
GESTATIONAL DIABETES
MELLITUS
IDENTIFICATION DATA
Name: -SHABANA
Bed no.: - 14
Address: -H no.-331, Gyan Mandir Road Near Madina Masjid Badarpur, New Delhi
Gravid: -G₂P₁L₁
Diagnosis: - G₂P₁L₁ with 36+4 weeks with GDM with single high BP reading
SUBJECTIVE DATA
1. ADMISSION HISTORY: Patient having pain complaints since night then she went to badarpur PHC, they
checked B.P which comes out 160/96 mm hg from there they sent mother to safdarjung hospital. She
had her GCT reports with 199mg/dl done on 25/1/2017
She also have complaints of mild breathing discomfort.
2. PERSONAL HISTORY: She is married for 2yrs . They live in a rented house paying Rs. 1500 for rent, living
in a single room having kitchen in same room.
She is a non-vegetarian and has a normal bowel pattern. No habit of tobacco chewing or smoking. She
has normal sleep pattern of 5-6 hrs a day.
3. MEDICAL HISTORY: Patient have No history of T.B, HTN. No history of any skin infection.
5. FAMILY HISTORY: Shabana and her husband lives in a nuclear family in Delhi. They have one daughter
of 3yrs of age. Her husband is the only earning member. Rest of the family members live in village.
Shabana’s mother had DM and HTN. Rest of the family members have no history of T.B., DM, HTN and
epilepsy.
6. MENSTRUAL HISTORY: She attained her menarche at the age of 13 years. She had regular cycle of 28-
30 days and has a regular blood flow for 4-5 days, associated with mild dysmenorrheal. No history of
oligomenorrhea, menorrhagia, etc.
L.M.P.-22/06/2016
E.D.D.-29/3/2017
7. OBSTETRICAL HISTORY: She is G₂P₁L₁. She have one girl child before 3yrs. Previous delivery was in the
hospital only with no complications. Child was active and healthy.
8. HISTORY OF PRESENT PREGNANCY: 1st TTIMESTER- During her first trimester, she had mild nausea and
vomiting in morning that got relieved on its own. She was given 1 st dose of T.T after one month
received the second dose of T.T.
2nd TRIMESTER- Her ultrasound was done on 19/11/2016. Findings were SLIUF of 19+4 weeks,
placenta was found anteriorly low lying . She was advised to take maximum rest at that time.
3rd TRIMESTER- She also had an ultrasound on 29/01/2017. Findings were SLIUF of 28+3 weeks and
placenta lower end was found well above the internal os. EFW= 1.26gm. her GTT was done on
6/02/2017 and results was 219 mg/dl. 4Hourly monitoring of blood glucose was advised.
9. CHIEF COMPLAINTS OF PRESENT PREGNANCY – Mild pain in abdomen, frequent urination, fatigue,
cough and dry mouth, dry itchy skin.
OBJECTIVE DATA: Patient is looking anxious. She is having frequent urination.Urine output was 2700ml/
hr Her RBS reading were recorded as:
date BBF ABF BL AL BD AD
6/02/2017 75 88 84 113 110 132
7/02/2017 87 106 72 172 92 86
8/02/2017 73 83 76 158 99 112
TREATMENT GIVEN:
o Diabetic diet
o T. amoxyclav 625 mg TDS
o Syp. Grillinctus 2 Tsf TDS
o Steam inhalation TDS
o Tab. FS/OC/BC
PHYSICAL EXAMINATION
General Appearance :Normal built, General condition is fair, hydration is adequate, looks tired,
tonsilitis
Breast : Soft, secreting colostrums seconsary areola visible, nipples are normal, no
Chest : Heart sound S1 & S2 is heard, lung sounds are normal and no evidence of
Wheezing or crackles.
Abdomen : INSPECTION: Linea nigra and striae visible in lower abdomen, abdominal girth – 40 cm ,
PALPATION: Fundal height – 36cm, height of uterus – 34 weeks, uterus is relaxed, active
fetal movements present, fetal parts felt, Auscultation: 146/min
GI system : normal intake of diet, bowel movements are normal, sometimes constipated
Bleeding per Vagina : Nil
INVESTIGATIONS
DATE INVESTIGATIONS REORT NORMAL VALUE
6/02/2017 -GCT 199Mg/dl Less than 140mg/dl
-TSH 3.o5 0.3-3µg/l
-HIV Non-reactive Non-reactive
-HbsAg Reactive Non-reactive
-VDRL Non-reactive Non-reactive
7/02/2017 -Hb 11.3gm/dl 12-14gm/dl
-TLC 14000/mm³ 5000-10000/mm³
-PLT 198000 lakh 1.5-3.5 lakh
-S. bil 0.41mg/dl 0.1-1mg/dl
-RBS 114mg/dl 80-120mg/dl
-KFT
Blood Urea 12.6mg/dl 10-40mg/dl
Serum Creatinine 0.8mg/dl 0.6-1.2mg/dl
Uric Acid 4.0mg/dl 3.5-5.5mg/dl
Anti-HbsAg Negative Negative
Urine Routine normal normal
8/02/2017 -Sputum culture Negative Negative
-fundus NAD Normal
-HbA1c 7.2 <7
-Glucose tolerance test
FASTING 138mg/dl 105mg/dl
1 HOUR 222mg/dl 190mg/dl
2 HOUR 186mg/dl 165mg/dl
9/02/2017 -Hb 11.8gm/dl 12-14gm/dl
-TLC 12000/mm³ 5000-10000/mm³
-PLT 190000lakh 1.5-3.5 lakh
-S. bil 0.21mg/dl 0.1-1mg/dl
-FBS 78mg/dl 70-110mg/dl
-T. protein 7g/dl 6-8g/dl
-albumin 4g/dl 3.5-5g/dl
-KFT
Blood Urea 14.8mg/dl 10-40mg/dl
Serum Creatinine 1.1mg/dl 0.6-1.2mg/dl
Uric Acid 3.2mg/dl 3.5-5.5mg/dl
On 6/02/2017, ultrasound was done, reports showed SLIUF in cephalic presentation, cardiac activity present,
placenta-fundo anterior , extending to rt lateral, Liquor-adequate, FL- 33wks 4 day.
ABOUT THE DIAGNOSIS
Gestational diabetes (or gestational diabetes mellitus, GDM) is a condition in which women without
previously diagnosed diabetes exhibit high blood glucose (blood sugar) levels during pregnancy (especially
during their third trimester). Gestational diabetes is caused when insulin receptors do not function properly. This
is likely due to pregnancy-related factors such as the presence of human placental lactogen that interferes with
susceptible insulin receptors. This in turn causes inappropriately elevated blood sugar levels.
Gestational diabetes generally has few symptoms and it is most commonly diagnosed by screening during
pregnancy. Diagnostic tests detect inappropriately high levels of glucose in blood samples. As with diabetes
mellitus in pregnancy in general, babies born to mothers with untreated gestational diabetes are typically at
increased risk of problems such as being large for gestational age (which may lead to delivery complications),
low blood sugar, and jaundice. If untreated, it can also cause seizures or stillbirth. Gestational diabetes is a
treatable condition and women who have adequate control of glucose levels can effectively decrease these risks.
The food plan is often the first recommended target for strategic management of GDM.
Women with unmanaged gestational diabetes are at increased risk of developing type 2 diabetes mellitus (or,
very rarely, latent autoimmune diabetes or Type 1) after pregnancy, as well as having a higher incidence of pre -
eclampsia and Caesarean section; their offspring are prone to developing childhood obesity, with type 2 diabetes
later in life. Most women are able to manage their blood glucose levels with a modified diet and the introduction
of moderate exercise, but some require antidiabetic drugs, including insulin.
INCIDENCE: Gestational diabetes affects 3-10% of pregnancies, depending on the population studied.GDM
rates are increasing and currently affect between 7% and 18% of pregnancies in the U.S. Increases in GDM rates
may result in a greater overall population with obesity and type 2 diabetes . GDM rates vary considerably by
race & ethnicity
PATHOPHYSIOLOGY: The precise mechanisms underlying gestational diabetes remain unknown. The
hallmark of GDM is increased insulin resistance. Pregnancy hormones and other factors are thought to interfere
with the action of insulin as it binds to the insulin receptor. The interference probably occurs at the level of the
cell signaling pathway behind the insulin receptor. Since insulin promotes the entry of glucose into most cells,
insulin resistance prevents glucose from entering the cells properly. As a result, glucose remains in the
bloodstream, where glucose levels rise. More insulin is needed to overcome this resistance; about 1.5-2.5 times
more insulin is produced than in a normal pregnancy.
Insulin resistance is a normal phenomenon emerging in the second trimester of pregnancy, which in cases of
GDM progresses thereafter to levels seen in a non-pregnant person with type 2 diabetes. It is thought to secure
glucose supply to the growing fetus. Women with GDM have an insulin resistance that they cannot compensate
for with increased production in the β-cells of the pancreas. Placental hormones, and to a lesser extent increased
fat deposits during pregnancy, seem to mediate insulin resistance during pregnancy. Cortisol and progesterone
are the main culprits, but human placental lactogen, prolactin and estradiol contribute, too. Multivariate stepwise
regression analysis reveals that, in combination with other placental hormones, leptin, tumor necrosis factor
alpha, and resistin are involved in the decrease in insulin resistance occurring during pregnancy, with tumor
necrosis factor alpha named as the strongest independent predictor of insulin sensitivity in pregnancy. An inverse
correlation with the changes in insulin sensitivity from the time before conception through late gestation
accounts for about half of the variance in the decrease in insulin sensitivity during gestation: in other words, low
levels or alteration of TNF alpha factors corresponds with a greater chance of, or predisposition to, insulin
resistance or sensitivity.
ETIOLOGY
IN BOOK IN PATIENT
Positive family history (parents or siblings) Mother had diabetes
Previous birth of an overweight baby of 4kg or more
Previous stillbirth with pancreatic islet hyperplasia
Presence of polyhydramnios or recurrent vaginal
candidiasis in present pregnancy
Age over 30 years
Obesity
Ethnic group(east asian, pacific island ancestry)
SYMPTOMS
IN BOOK IN PATIENT
Blurred vision Vision is normal
Fatigue Patient looks tired
Increased thirst Patient is having dry mouth and
Frequent infections, including those of the bladder, increased thirst
vagina, and skin Not present
Increased urination Having frequent urination
Dry skin Patient is having dry skin
Nausea and vomiting Nausea and vomiting present in first
Weight loss despite increased appetite trimester
Increased appetite present
DIAGNOSTIC FINDINGS
IN BOOK IN PATIENT
Symptoms of ppolyuria, polydipsia, polyphagia Present in patient
During labour
Prolonged labour: due to big baby
Shoulder dystocia: due to disproportionate growth with increased shoulder/heart ratio
Perineal injuries Post partum haemorrhage
Operative interference
During puerperium
Puerperal sepsis
Lactation failure
FETAL
Fetal macrosomia
Congenital malformation
MANAGEMENT
IN BOOK IN PATIENT
Dietary management On diabetic diet, diet chart given restricting
Regular blood sugar charting diet to 2000kcal
Exercise 4 hrly blood sugar charting is done
Human insulin to be started if plasma Patient is advised to walk daily
glucose level exceeds 90mg/dl and post Patient is getting 2 units Regular insulin bbf,
prandial glucose more than 200mg/dl before lunch and before dinner
Usg to be done for fetal well being and to Usg was done, liquor was adequate and EFW
assess fetal size was normal
HbA1c, glycoalbumin, blood
biochemistry, peripheral
blood in general: measured once per
month. HbA1c doen – 7.2
Urine ketone bodies, protein, Urine ketones are negative
qualitative measurementof urinary Nst done –non reactive
glucose: twice per week
Non-stress Test (NST) to be done weekly
afert 30 weeks
NURSING MANAGEMENT
Nursing Diagnosis
Nursing diagnosis : Imbalance nutrition: less than the body requirement related to inability to utilize
nutrients (imbalance b/w intake utilization of glucose)
EVALUATION OF GOAL
Patient is maintaining a balanced diet by following the diabetic diet.
Patient is doing walk after meals
Nursing diagnosis Risk for fluid volume deficit related to loss of fluids from the body due to polyuria
EVALUATION OF GOAL
Patient is maintaining the hydration status.
-vitals are stable.
Nursing diagnosis Activity intolerance related to increased energy demands and diseased condition
EVALUATION OF GOAL
Patient is able to do do self activity, not so dependent.
-takes brisk walk and takes adequate rest.
.
DRUG STUDY
Tab osteocalcium Calcium supplement Tablet osteocalcium 500 Assess patients serum calcium level ,
mg p.o o.d for 6 weeks so as to get the baseline data .
HEALTH EDUCATION
1. NUTRITION
Advised the patient to eat well bal anced diet.
It should be highly nutritious and easily digestible.
Diet with 2000-2500Kcal/day for normal weight woman and restriction to 1200 -1800kcal/day for
overweight woman is recommended.
Advised for high protein diet.
Advised the diabetic mother to avoid foods like sweets, honey, sugar, cakes, fried foods, potatoes,
cold drinks etc.
Advised to avoid food rich in fat and calories.
Advised to take plenty of fluids (sugar free).
Also advised to take meals after 15 min of insulin administration
Curd 150
Salad - 100 gm
3 pm: Fruit -100gm
EVENING time(5pm): soup /tea (sugar free)
DINNER: Roti(wheat) – 2 piece
Green vegetable – 200 gm
Salad - 150gm
Curd/paneeer – 100gm
Dal – 50gm
WHAT TO EAT
- Loki, lady’s finger, Tinda, Bitter gourd, raddish, kheera, onion
- Apple, guava, papaya
- Brinjal, cauliflower, bitter gourd-soup
- Milk (double toned) – 500m (milk/curd/tea)
- Paneer – 60gm/ egg (only white)
- Cereals + pulses – 150gm
- Vegetables, fruits – 100-200gm
- Oil – 6 small tsp
- Salt – half tsp; water
2. PERSONAL HYGIENE
- Maintain personal hygiene byy daily bath, combing, dress change
- Avoid exposure to infection, avoid crowds.
3. REST
-TAKE REST : Sleep for 2-3 hrs in afternoon and 8-10 hrs at night
-Avoid remaining in one position
- Avoid strenuous work
5. COMFORT MEASURES
- Wear comfortable clothes, wear loose clothes and avoid tight clothings.
- Avoid travelling in 1 st and 3 rd trimester
- Avoid long hours of standing or sitting in one position.
- Avoid coitus during 1 st and 3 rd trimester.
6. MEDICATION
- Taught methods about the method and techniques of injecting iinsulin; the dose, route and side effects.
- Advised to do self glucose monitoring before insulin administration.
- Advised to have meals atleast after 15 min of insulin administration.
- Advised patient to report any side-effects.
CONCLUSION
Gestational diabetes mellitus is one of the leading cause of perinatal mortality. Early pre -conception
councelling and good antenatal care can prevent the complications and death of mother and fetus.
BIBLIOGRAPHY
D.C. DUTTA, TEXTBOOK OOF OBSTETRICS, HHIRALAL KONAR, 7TH EDITION, PG NO,- 281-291
AMY. M. KARCH, NURSING DRUG GUIDE, LIPPINCOTT (LWW) 2011, PG NO: 641, 703,