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VILLARAMA
NEONATES (0-28months) 02 Precaution:
IMMEDIATE DELIVER ROOM CARE: 1. NB: Retrolental Fibroplasia
ESSENTIAL INTRAPARTAL NEWBORN 2. PRE-TERM: Retinopathy of Prematurity
CARE (the fragility of the blood vessels of the
In support of the Millennium Goals 4 2015 retina)
Sustainable Development Goal: Good Health
and well-Being for People TO REDUCE *CYANOSIS: the more crying, the bluer he gets:
CHILDHOOD MORTALITY TRANSPOSITION OF THE GREAT VESSELS
*02 Mask (5L per minute): if the 02 sat dropped 70% and
NURSING RESPONSIBILITIES CENTRAL CYANOSIS
A AIRWAY *15 minutes – increase the O2 saturation by 5%
B BODY TEMPERATURE *Pulse Oximeter: Heel or Great Toe (Bigger baby)
*Ambient (Environmental) Oxygen: 21%
C CERTIFY THE BIRTH
D DETERMINE ADAPTATION TO EXTRAUTERINE LIFE -
BODY TEMPERATURE TIME BOUND/
APGAR;
BAND INTERVENTION:
INTRAUTERINE: BPS
*Air-conditioned Room: infection control and also machines that
AIRWAY may overheat
TIME BAND/ BOUND INTERVENTION: Physiological heat loss after birth: 37.2 C down to 35.5
TIME BOUND – priority intervention to 36.5 C
Umbilical Cord: “Daluyan ng Buhay” Maximum is 35.5, anything lower causing EXTREME
HYPOTHERMIA can cause COLD STRESS causing
Prevent Asphyxiation: Accounts 31% of NB death
ACIDOSIS
Prevent Aspiration
PRONE TO COLD STRESS DUE TO HYPOTHERMIA
ASPHYXIATION EXTREME HYPOTHERMIA
Umbilical Cord is clamped
↓ NON-SHIVERING Burns Brown Fats for heat: Not
No more 02 from placenta THERMOGENESIS CHO or CHON since there is
↓ no source
Hypoxia (decreased O2 in tissue) Think about when the person These are the supports of the
↓ shivers takes energy with every organs since ligaments are not
Hypercapnea (increased CO2) shake, what more to a baby yet mature
↓ without glucose
Acidosis: Buffer HCO3, Respiration, Urination
↓ Increases O2 consumption Ketones/Fatty Acids: The by-
CNS Depression: Respiration is depressed or product of the Fat Metabolism
slowed down; Kussmaul’s Breathing, LOC goes to
comatose state Decrease O2, Increase CO2 ACIDOSIS
ACIDOSIS
oxia – tissue
emia – blood EARLIEST SIGN OF CNS DEPRESSION
Acidosis kills faster than Alkalosis NEONATES: cannot be woken-up
7.35-7.45 – base NB: Glucose is stored in the liver; 36-39th week, baby stores fat
6 lower – acidosis term baby bounces stores CHO neonates normally have
7 above – base hepatomegaly
Every time you urinate – you bring out acid
SPONTANEOUS WAKING: Hunger or feeling of
Neonates must breathe after birth; If not discomfort (position or wet diapers)
ASPHYXATION may result (hypoxia, Hypercapnea, NEONATES WITH HEPATOMEGALY: they have
Acidosis) an extra supply of glucose for the next few days;
A Crying Baby is a Breathing Baby Mother’s Milk production is still meager; They
Neonates breathes after birth by crying so stimulate the would rather rest than to eat.
NB to cry effectively after birth to prevent asphyxiation Do not push yet if not more than 4 cm
SUCTION WITH A BULB SYRINGE (unlikely to hit the o PANTING and with CLEANSING
carina) PRN if (with Nasal Obstruction/Meconium BREATH (to relax the uterus)
Stained amniotic fluid) TO PREVENT ASPIRATION o Improper pushing leads to a neonate
Amniotic fluid is normal to be in the lungs because it with a LONG HEAD due to prolonged
will be absorbed by the pulmonary tissues labor
TO PREVENT ASPIRATION – turn the baby to lateral If the baby is not crying well: CONGENITAL
position HYPOTHYROIDISM, CONGENITAL CRETINISM
TRACHEOESOPHAGEAL FISTULA/ ATRESIA: There (one reason)
is mucous even in esophagus not only in lungs ONLY PUSH WHEN: [1] Full dilated; [2] Dilated
DANGERS OF SUCTIONING: VAGAL STIMULATION and Efface
bradycardia; reducing 02 reserves Pelvic Dystocia: the head is in the inlet but the
Encourage to cry effectively to maximize lung shoulders have not entered the area
expansion
ACYANOTIC CYANOTIC OXYGENATOR): oxygenate the blood and bypass the heart; Also
Increased Obstruction to Decreased Mixed blood induce Hypothermia (slows down oxygen demand of all the
pulmonary blood flow pulmonary flow organs) using Hypothermic blanket and tuck the patient with ice;
blood flow from the blood flow Given PLEUR VAC to drain the secretion or CHEST TUBE to
ventricles restore negative pressure
VSD COA TOF TOGV
ASD AS TA TA MEDICAL MANAGEMENT
PDA PS TAVR OBJECTIVE 1: PREVENT CHF
AV CANAL HLLS CHF MANAGEMENT
Drugs: Digoxin and Diuretic
CONDITION
SYMPTOMS
ACYANOTIC
Breathing
CYANOTIC
Central Cyanosis
D Diet: Decrease Cardiac Demand
Decrease occurrence of infection
Difficulty Clubbing of fingers o Improve the CO: Give DIGOXIN (Cardiac
COMPLICATION CHF Clot (Thrombus) Glycoside): Increases strength of contraction
Cerebral Thrombosis o COLORED FOOD FOR DENGUE: The color
MANAGEMENT Digoxin Prevent “TET” spells change is not for the stool but for the vomitus
Diuretics Polycythemia that may mask the bleeding
Diet – Low monitoring o Prevent Na retention and promote elimination
sodium Phlebotomy of excess fluids (FUROSEMIDE: Diuretics);
Decrease O2 Position – Squat/Knee SPIRINOLACTONE: takes a longer (2 days)
demand Chest effect so give the faster one and simply give
Provide O2 potassium supplements; measure the intake
and output (weigh diaper); Weight is the best
ACYANOTIC HEART CONDITIONS indicator of water loss; Weigh the patient OD;
Congestion of the heart chambers and causes the heart Monitor potassium (3.5-5.5 mEq/L); Replace
to compensate by increase rate if contraction leading to K loses (SAFEST: Natural; GIT [fresh fruits,
CHF: fruit juices/ if oral do not crush time released
CC: Early Pulmonary Symptoms: Dyspnea, fast Kalium Durule]; IV potassium is incorporated
breathing, moist cough, rales/ crackles into a side bottle (piggy black) and with a
main line, NEVER PUSH!); Observe in K
DX: CXR: pulmonary edema and cardiomegaly; but
administration (monitor the ECG; every 15
HEPATOMEGALY could be seen in Cardiac
conditions; 2D-ECHO (echocardiography) & MRI: minutes)
identifies the type of the heart o Give ACE inhibitors
SURGERY: PH SOCIETY OF PEDIATRICIANS: After o Low Sodium intake: Low sodium formula
5th birthday (the baby will now go to school and (LONALAC), Clarification on solids allowed;
increased activities; able to safe for funding) Not only the salty foods are rich in sodium
CARDIAC CATHETERIZATION: Identifies pressure (eg Cola [HC03]; breads [HC03]; Tocino
inside the heart (CHF); Also interventional which [Sodium Nitrate])
inserts a stent; the femoral is insertion site;
immobilize the legs (Sand Bags or let the mother MONITOR CR/HOLD IF SLOW
hold), bed rest, Assessment of the site (Circulatory Less than 1 Less 100
problems: clots will form, determine for COOL skin 1-5 y/o Less 80
then check for pedal pulses) 6-10 y/o Less 70
11&above Less 60
RIGHT SIDED FAILURE LEFT SIDED FAILURE EARLIEST SIGN OF TOXICITY:
SYSTEMIC PULMONARY GIT symptoms; M/V, vomiting, Anorexia, Abdominal
CONGESTION CONGESTION Pain, Diarrhea
GIVEN BEFORE MEALS: on an empty stomach; Not
Distended neck veins, pedal Dyspnea, rales, productive due to absorption (for most medications this is the
edema, ascites, hepatomegaly cough, rales/cough, pulmonary reason) so that you determine if the problem is caused
edema, FROTHY THICK by the toxicity or the effects of eating being full.
SPUTUM (blood tinged; pink;
rupture of alveolar capillary *IV PEDIA: usually peripheral or in central line
membrane) *IV will burn or turn black in excess potassium
*FIRST SIGN OF HYPERKALEMIA: T-wave peaks (for
*2D echo and cardiac Catheterization: the means to determine ECG); GIVE GICKS: the insulin will trap the potassium into
the intervention the the cell
*Normal Cardiomegaly: Old Age & Sports *Added salt in cooking must be a pinch or at the rate amount to
pulmonary circulation; Connection from the aorta to the
pulmonary artery
FORMEN OVALE: Also to bypass the pulmonary
circulation
P PENICILLIN
PHLEBOTOMY
PROMOTE Fluids
o Decease 02 Demand
CLOSURE in fetal circulation:
o PROPRANOLOL to decrease TET SPELL,
o FUNCTIONAL
Prophylactic antibiotics for bacterial
It will stop functioning and will
endocarditis
close; The lungs will expand, 02
o Monitor Hgb and Hct count: detects early
level in the body; Now the pressure
POLYCYTHEMIA
from the right shift to the left
o ASSISTS in Phlebotomy: blood letting and
caused by pressure from opening
replace with Plasma of the same amount to
of the lungs; The FORAMEN
make the blood thinner
OVALE (2-3 months is Anatomical
o Increase Fluid/ Maintain IVF line as
Closure) will now close.; the
necessary: Water games (Water guns for
increased oxygen will increase 02
feeding; Grapes oranges, Watermelon;
levels and collapse the PATENT
Jellow)
DUCTUS ARTERIOSIS (3-4 weeks
o Positioning during attacks: allow to squat or
or a month is anatomical closure)
knee-chest, give 02
o ANATOMICAL
o Monitor activity tolerance/ LOC: Hypoxic
Foramen Ovale: Not cyanotic but
effect; Initially restless; Decreased 02, the
may become dyspnic; The heart in
CHILD is hard to awaken / aroused
this condition will become
Pain and Hunger: This will wake up a client
congested at the RA since the
shunting from the left side, this will
TRANSPOSITION OF THE GREAT VESSELS
trigger a compensating mechanism
by contracting faster; The over- Most dangerous; Presents at the moment of birth
contraction will cause the heart will “The Harder they cry, the Bluer they get”
get tired and the quality of ONLY HOPE: that the baby has septal defects;
contractions diminished, Low DUCTUS ARTERIOUSUS: if open, the force of the LV
stroke volume = CHF will push the oxygenated blood towards the Aorta
Difference to CHF for Pedia is: (systemic circulation)
Streptokinase (Thrombolytics), S/SX: Persistent Cyanosis In spite of vigorous crying;
Aspirin, Low fat (Anti Lipidimics); hypoxia in spite of oxygen therapy; Less symptomatic if
Same DIGOXIN, DIURETICS, with septal opening
BETA BLOCKERS MGMT: Give prostaglandin E2 to open ductus
o STARLING’S RULE: the stretches will dictate arteriosus; PALLIATIVE : Baloon Septostomy;
the contraction of the heart CORRECTIVE: Open Heart Surgery, MILLER
RASHKIND PROCEDURE
VSD Prostaglandin is a muscle and BV relaxant; This will
Same as ASD cause the relaxation of BV and keeps the defects open
Only in the ventricles *INDOMETHACIN (Prostaglandin Inhibitor): will
close
PATENT DUCTUS ARTERIOSUS
The RV is now over stretched since the blood returns TETRALOGY OF FALLOT
from the aorta into the heart; D Displaced Aorta
R Right Ventricular Hypertrophy
COARTATION OF THE AORTA O Opening in the septum (VSD)
Descending aortic stenosis, now the LV now gets P Pulmonary Artery Stenosis
congested; THE BP becomes HIGH: Higher in the Pressure on the RIGHT side is higher than left
ARMS (upper Body) than the legs (lower body) May go home or may not diagnosed at birth since S/SX
*Normally: the legs are higher in pressure due to is not so evident; When he cries the baby turns blue but
distance, diameter of the vessels and pressure when relaxed turns pink;
from the heart. S/SX
*For the legs: Add the BP +20 to the Systolic and o As the baby active, the S/SX gets more
Diastolic pressure pronounced because of increase oxygen
supply or TET SPELLS (you are kinking or
occluding the vessels in the legs
J.E.A.D. 11 UST CON BATCH 2018
PEDIATRIC NURSING – R. VILLARAMA
o The decreased pressure in on the right side o Cover the sac with sterile gauze (HOLD THE
of the heart due to return from circulation DRESSING SARAN/ CLING WRAP) with
o TET spells will improve circulation); perform
Shamroths
o Boot Shaped Heart (due to cardiomegaly) HYDROCEPHALUS
TET SPELL: if can stand (Toddler) SQUAT if Infant Hydrocephalus enlarged ventricles brain pushed
Knee Chest against the cranium increased pressure baby still
Clubbed fingers due to peripheral hypoxia; has fontanels, can expand able to accommodate the
POLYCYTHEMIA due to chronic Hypoxia (Blood swelling
clotting due to increased BLOOD products in small BV: May be caused by a tumor
Cerebral Thrombosis [RARE in pediatrics]) Complication of encephalitis
Stunted physical growth and delayed development Bulb-shaped head, Sunset eyes, Distended scalp veins;
High pitched cry, Increased ICP (Projectile vomiting)
NEURAL TUBE DEFECTS Give morphine to decrease pain
CAUSE: inadequate intake of FOLIC ACID during VS: Increased Systolic and normal diastolic (widened
pregnancy; Not enough folic acid pulse pressure); TEMP (variable, high or normal but
Posterior lamina is missing or absent contents went does not go down); RR low; HR low
out TRANSILLUMINATION: placing a strong flashlight by
85% of children with neural tube defects develop the head to see the fluid in the head
HYDROCEPHALUS More dangerous for adults since there are no fontanels
Drugs, radiation MANAGEMENT
Sources of Folic acid o Position side-lying (especially if opisthotonic:
o Green leafy vegetables o Nuts arched bad)
o Legumes o Assessment of signs of increased ICP:
o Brown rice measure (Earliest sign is Altered LOC:
o Strawberries restless, sleepy)
Restlessness; for hypoglycemia
SPINA BIFIDA OCULTA while ALOC is increased ICP, in
Hidden; no sac pedia
Usually not visible externally o Dimple at sacral area o HCOD (head circumference of the day)
With hair o Measures to prevent increased ICP
No intervention necessary SURGICAL MANAGEMENT
o Ventriculostomy to relieve pressure
SPINA BIFIDA CYSTICA o Insertion of shunt to bypass the point of
With sac obstruction
Intervention is necessary So CSF can be recycled: Drainage
MENINGOCELE- consist of sac-like cyst of meninges
filled with spinal fluid reabsorbed by the blood vessels
MENINGOMYELOCELE: protrusion of a sac-like cyst mesenteric vessels (the CSF is a
containing meninges, spinal fluid and spinal cord with good source of glucose)
its nerves. Paralysis of lower extremities. Clubbed foot- o Shunt Revision- based on the length of the
talipes equinovarum. tube inserted in the body (scheduled)
ENCEPHALOCELE: Herniation of the brain and o Obstruction of catheter (unscheduled shunt
meninges through a defect in the skull - most revision); this happens when the S/SX
dangerous returns or Increased ICP
o Medications are not effective
ASSESSMENT
o In adults, permanent shunting is done with:
o Observe for movement of the lower
Removal of brain lesion
extremities, apply pain;
Clipping of aneurysms
o With movement: meningocele (+) talipes
o Post-op Nursing Care of Shunt Insertion:
equinovarus (clubfoot) - meningomyelocele
Routine post-op VS monitoring
OVERALL GOAL
Position: FLAT on the unoperative
o Protect the sac against pressure, injury and
side to prevent pressure on the
infection
shunt valve and too rapid drainage
MANAGEMENT and reduction of CSF that may
o Surgical closure preferred within 24-48 hours cause subdural hematoma
after birth to prevent local infection and Do not carry the infant
trauma to the exposed tissues Monitor for increased ICP
o Prone position Observe for abdominal distention
o Bladder cannot expel all the contents due to (peritonitis or abdominal ileus)
decreased innervation to the detrusor
muscle: Catheterize every 4-6 hrs GASTROINTESTINAL
(Intermittent catheter procedure; straight CLEFT LIP AND CLEFT PALATE
catheter; high risk for infection if retention is
Cleft Palate: MC in males
still there)
Cleft lip: MC in females
o CERDÉ: Manual Compression from Umbilical
to symphysis pubis Vitamin A, Intake of Anticonvulsants, Hereditary
o by BUT don’t Can cause problems with airway
put pressure on the sac Cleft palate: speech, defects in ear, hearing problem
o You can use diapers but don’t put it on SURGERY is a PRIORITY
o No problem with breastfeeding Will have coping problems as they grow up
LEUKEMIA
Most common form of childhood cancer
Malignant disease of the bone marrow and the
lymphatic system