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Healing Skin by altering it’s Select Category

pH & microbial
(Malassezia) pro le
Skin conditions
such as Psoriasis,
Eczema,  atopic
dermatitis,
Dandru , dry
skin, Tinea
Versicolor,

Rosacea,  Seborrhoeic dermatitis, Acne are in many cases thought to


be caused by the fungus Malassezia.

Few people know this but natural healthy skin has a pH around 4.


That is quite acidic and actually a good thing. Acidic pH helps maintain
skin integrity and keeps bene cial ora attached to the skin and bad
ora such as fungus away. (ref.)

Malassezia grows optimally in an environment when the pH is


between 4 and 8. (ref.) Therefore it is critical the skin pH be around 4
or ideally slightly lower.

The use of soaps, water, creams and makeup on the skin can alter it’s
pH considerably. Washing skin with soap and water may change the
skin’s pH by up too 2 points making it more alkaline and it may take
over 6 hours to return back to normal baseline. Many manufacturers
know this and thus “pH balance” soaps, lotions and creams to make
them more acidic. And in ignorant cases more alkaline, so it’s prudent
to check.

Malassezia is a lipophilic yeast meaning it eats fats and oils to make


itself grow. In a lab environment olive oil is used to enhance the
growth of the fungus. (ref.) 
***Therefore many lotions, creams and oils used to hydrate and
moisturize the skin will actually make the problem much worse
despite o ering temporary relief. Using most of them is like
adding fuel to a re!*** 

Most species of Malassezia consume fatty acids with a carbon chain


length of 11 through 24.  (ref.)

Therefore you want to check every single ingredient in a lotion/cream


to make sure it is not within that range or else it will feed
the Malassezia. For a list of oils (12-24) to avoid click here for a table.

Ointments that appear to be safe are:

Aquaphor is 41% petrolatum, which is composed of very long


carbon chains of C25 to C30
Petroleum Jelly has carbon number mainly higher than 25 (ref.)
MCT oil has carbon chain lengths of 8-10

Another reason olive oil is a bad for example, is that it


contains  Tyrosol which has been found to be a quorum-sensing
molecule that accelerates the growth of the fungus Candida albicans
which shares many similarities with Malassezia. (ref 1) (ref 2) 

Even if the skin issue is not caused speci cally by  Malassezia, it may
be caused by another bacteria. The following strategies will still be
e ective however minus the pharmaceutical antifungals. Bacteria also
form bio lms, and the pH component is critical in mediating the entire
skin microbiome.

Short Term Fix:

There are several strategies that can be used to kill the fungus and
provide temporary relief. Keep in mind however it will grow back
quickly unless the underlying environmental pH conditions skin are
changed from the inside out.

1. Breakthrough Malassezia bio lm
Malassezia as it grows forms a bio lm around itself for protection
purposes. Pharmaceutical antifungals can be used but often have
limited impact and will need to be used for extended periods of time
due to the very slow process of moving through the bio lm.

Bio lm on Skin

A faster strategy perhaps is to use bio lm busting compounds. Some


possible compunds are:

Xylitol – reported by many people to be very e ective, try this


rst
Candex formula applied topically (1 anecdote review here)
Chitinase because Malassezia cell wall is made up of chitn
Fibrin enzymes such as Serratiopeptidase and nattokinase may
also work
Aloe Vera appears to also dissolve bio lms (reference needed)
Chitosan (source)

2. Kill the fungi

Pharmaceutical fungicides include:

Climbazole  *considered the best found in “Hegor 150”


Clotrimazole
Itraconazole
Ketoconazole, Piroctone Olamine, and Lotrimin Ultra

3. Create a temporary hostile (acidic) environment so it does not grow back

Acidifying the skin topically using Apple Cider vinegar has also helped
many people. Other acidic compounds may work as well. Lactic Acid
and urea are often used in creams speci cally for eczema.
Sandalwood oil which contains the compound Farnesol may also be
helpful. Farnesol is a quorum-sensing molecule which inhibits the
growth of fungi such as Candida. There is some evidence to suggest
that it works on Malassezia as well. 

Lactoferrin inhibits adhesion (ref.)

An ideal topical lotion might contain….

Aloe Vera gel


MCT oil
Xylitol
Lactic Acid, Acetic Acid & Citric Acid
Climbazole
Sandalwood oil (source)
Niacinamide (B3), plus dl-panthenol (B6) and glycerin to
improve skin barrier
have a pH of 3.5 – 4

Places to buy ingredients:

http://www.modernistpantry.com/

Long Term Fix:

The primary root cause of the above listed skin issues is in most cases
the original use of antibiotics. Antibiotics even used once cause mass
extinction on the gut biome level permanently altering the ecology
which helps regulate many important functions in your body.
Antiobiotics are targeted towards bacteria and in the vacuum created,
often fungus take over. The goal here is to re-balance the gut biome
to a ect the skin. A person can a ect dramatic change when using
speci c pre and pro biotic strategies as has been demonstrated
recently with the wide use of Resistant Starch.

The most dominant variable governing skin pH appears to be lactic


acid. (reference) The skin has what is called an “acid mantle” that
protects against foreign invaders and helps maintain skin integrity.

So the goal here is to hopefully use the BEST pre and probiotics for
increasing lactic acid in the body. This appears to be best done by
supporting the growth of lactobacilli in the small intestine. Incidentally
this is same strategy to use to combat Candida growth in the small
intestine and in many cases it is probable that people have both
internal and external fungal infections concurrently. Over growth
of Malassezia on the skin is a good indicator of internal dysbiosis.

I have no idea if this strategy will increase lactic acid at the skin level,
however blood pH is tightly regulated (7.35 – 7.45), so I am hoping
lactic acid produced in the gut may transfer quickly to the skin if there
is an adequate supply.

Many studies and anecdotes seem to suggest that pre & pro biotics
can cure all manner of skin issues and I believe the lactobacilli
producing lactic acid are the primary reason for this. The problem is
results are often intermittent. For some people taking the probiotics
alone appears to be good enough. However in more serious cases it is
likely necessary to take a more aggressive approach especially if the
problem is systemic. How aggressive a person may need to be I don’t
know, it will be up to you to experiment.

How does skin become acidic? There appears to be several


mechanisms.

1. Lactic acid from microbes in the gut is taken up by blood and


then released through the skin (hypothetical)
2. Lactic acid byproduct from energy depletion in cells is excreted
through sweat
3. The conversion of phospholipids to fatty acids by enzymes in
the skin (source)

The goal is to maximally produce acetic, lactic & propionic acid


internally with the goal of having it secret through the skin. For a
prebiotic I recommend using pectin. In prebiotic comparison studies it
ferments all-around the best of many prebiotics with respect to lactic
acid producing bacteria.

The prebiotic Larch Arabinogalactan has been shown to dramatically


increase levels of Propionate. Propionic acid is known to be a very
potent fungicide. It is used commercially to control fungi in stored
grains and animal feed and has been tested speci cally on Candida
albicans for e cacy. (ref.)

Prebiotics recommended:

Apple Pectin, Acacia Senegal, Larch Arabinogalactan

Probiotics recommended:

L.Plantarum, Lactobacilli species

Acidifying Sweat:

Because tinea versicolor ares most frequently in hot climates when


sweating is increased it is likely that the neutral pH of sweat is what is
allowing the fungus on the skin to grow. Therefore by making the
sweat more acidic it’s possible to perhaps eliminate malassezia. The
following are some acids found in sweat that perhaps could be
boosted with supplementation.

Pantothenic acid aka. Vitamin B5 – ph of 4.41; is water soluble


 (also appears to help wound healing) (source)
Ascorbic acid aka. Vitamin C – ph of 4.10
Lactic acid
serine – ph of 2.21 as a carboxyl

Skin as the last line of defense – Possible Psychological


functions governing integrity of the skin boundary – a
Bodynamic theory

New Theory: (April 19, 2018)

There are at least two primary body-ego functions of skin


psychologically speaking and that is Energy Management and
Boundaries. Skin is the boundary layer between the physical self and
the environment serving the important function of di erentiation. At
an emotional level it is also the boundary which contains energetic-
emotional energy. Developmental disruptions in childhood and/or
trauma later in life can have a negative e ect on these important
psychological functions which then negatively a ect the endocrine
system that is responsible for regulating these psycho-physiological
parts of ourselves.

In the Bodynamic system there are four psychological boundary


layers:

1. Skin Boundary
2. Personal Space Boundary
3. Territorial Space Boundary
4. Social Space Boundary

The sensi-motor development of each boundary layer occurs during


di erent developmental time periods in relationship to the self and
people in the environment. I don’t want to get into the nuances of
what each boundary layer means (there is an e-course coming on the
subject soon), but the important point is that gaps in boundary
development and enforcement will put undue stress on other
boundary layers.

For example, a parent may not have respected a child’s personal


boundaries frequently overwhelming them physically or emotionally,
but may have respected their territorial space. In such a case
territorial space boundaries, keeping a living environment ultra
organized and clean, or alternatively strewn with personal clothes or
belongings is a way of using territorial boundaries to compensate for
a lack of ability in more intimate personal boundary space. Kind of like
a dog peeing to mark it’s territory.
If there is di culty in di erentiating personal, territorial and social
boundaries the last line of separation/defense/di erentiation
becomes the skin boundary. The ability to sense the skin boundary
and utilize it as a psychological resource is developed in utero to
about 1.5 years of age. Disruptions or trauma in this age range could
set up a person for having di culty for life if the body-ego is not
su ciently developed to allow regulation of this organ.

Energy Management is another Bodynamic Ego function that could be


at play here. Fascia which wrap around the muscles and are the layer
between muscle and skin are psychologically responsible for
containing high levels of energies and emotions.  Prolonged trauma or
stress can adversely a ect this important layer as it becomes over-
utilized in order to manage di cult emotions inside the self. This well
then have a negative e ect on the endocrine function in associated
areas.

Bodynamic bodymaping can be used as a tool to determine how well


the muscles and fascia are functioning from a psychological
developmental perspective. With future correlative research (which I
hope to personally engage in) it may be possible to establish causality
for the psychological reasons that underlie skin issues where there is
a breakdown in integrity and function. This may also help explain why
skin issues show up in di erent parts of the body, like the hands,
arms, face scalp for example. Muscles and fascia in each parts of
these body areas serve a speci c physiological and psychological
purpose.

The tricky thing here is that it may not be the a ected area where the
actual problem is. It may just be the area where there are actual
resources that are being over utilized and compensating for a lack or
resources elsewhere. This is why getting a full bodymap done, and
taking a look at the whole picture psychologically and physiologically
is important.

Old Theory: (Decreasing Sebaceous Gland activity – Psychological


endocrine factor – a Bodynamic theory)

It is well known that hormones (such as androgens  aka. “male


aggression energy”) a ect SG activity. Therefore emotional stress and
the physiological neural preferences could have an impact on
localized skin issues. See my bodymap for further analysis. Muscles
and fascia that are hypo-tonic signify areas of the body that are
pyscho-emotionally inadequately brain-neural mapped in the limbic
system. They are energetically under-resourced, and the lack of
containment and function, likely results in undesirable leakage of said
energy.

Sebum is a mixture of fats, and releasing to many be a sign of poor


energetic emotional containment. Emotions as opposed to thoughts
are denser energetically and are symbolized biochemically by fat,
where thoughts could be biochemically symbolized by glucose. The
skin ideally acts as a barrier to contain emotions. This is done
psychologically by sensing into the body and feeling emotional energy
bounded by the skin and contained by it. For some people this
skill/neural map may need to be learned, and it’s possible this may
have the e ect of decreasing hormonal activity at the skin level and
therefore SG activity reducing triglyceride leakage or unnecessary
expulsion.

Acidity could also be symbolized by aggression energy, and a lack of


containing and nurturing aggression energy could lead to an
alkaline/more permeable skin barrier. Aggression is an important life
force factor, one that too many people “leak out/expel” emotionally
due to fearing it’s power, or bad beliefs around it. New neural maps
will need to be created to harness the bene ts of aggression psycho-
somatically and hypo-tonic fascia and muscles need to be brought
back online, in order to a ect the endocrine-skin-barrier functional
system.

Reference:

The composition of Sweat, with special reference to the


vitamins
Analyzing and Mapping Sweat Metabolomics by High-
Resolution NMR Spectroscopy  (On all the surface parts of the
human body examined in this work, the main constituents
forming a sweat metabolic pro le are lactate, glycerol,
pyruvate, and serine.)

Additional Notes:

“It has been shown in vitro that glycine stimulates the fast
growth of M. furfur, and when this amino acid is exhausted,
yeast cells employ tryptophan as a nitrogen source, increasing
the production of indolic metabolites.” (source)
“This was based on the observation that medium-chain fatty
acids could delay the growth of the seven Malassezia species in
vitro…” (source)
“This study shows the e cacy of an exogenous NO-releasing
cream in treating tinea versicolor.” (source)
“The anatomical substrate of the epidermal barrier function,
which is defective in atopic eczema, is the stratum corneum of
the epidermis, a thin biological membrane that covers the
whole body surface. It is made up of the keratinized, terminally
di erentiated epidermal keratinocytes of the interfollicular
epidermis bound together by corneodesmosomes, lled with
natural moisturizing factor and embedded in a lipidic matrix
that is composed mainly of ceramides, cholesterol, fatty acids,
and cholesterol esters. The natural moisturizing factor is
formed by the degradation of llagrin, comprising substances
such as lactic acid, sodium pyrrolidone, carboxylic acid,
urocanic acid, and urea. Decisive for the proper function of
the stratum corneum is the maintenance of a pH gradient
between its acidic outer and basic inner surfaces that motors
many vital functions of this life-imperative biological
membrane.”  (source)
“those authors pointed out, linoleic, oleic, and palmitic acids,
which are potent growth stimulants of Malassezia
species” (source)
“The use of acidic topical preparations containing alpha
hydroxy acids, such as lactic and glycolic acid, for
kereatolysis has proved e ective in clinical practice. The
treatment of atopic dermatitis is aided by their pH regulating
properties.” (source)
“The e ects of acetic acid (AA) and hydrochloric acid on S.
aureus bio lm formation were evaluated in bullous impetigo
and pemphigus foliaceus isolates and were both shown to
decrease glycocalyx production. AA had a superior e ect in
reducing bio lm-associated S. aureus counts, indicating a
speci c e ect of AA unrelated to low pH. In the clinical setting,
AA 2.5% ointment was reported to reduce S. aureus counts in
pemphigus foliaceus lesions…” (source)
Meta analysis of lactic acid bacteria as probiotics for the
primary prevention of infantile eczema (source)
“The new antimicrobial hydrogel, made of 90 percent water,
gloops together spontaneously when warmed to body
temperature. It can bust through bio lms and kill a whole host
of bacterial types, from small bugs like E. coli to large bugs like
methicillin-resistant Staphylococcus aureus. The hydrogel is
comprised of specially designed polymers, which are
biodegradable and positively charged. When mixed with water
and warmed up, the polymers self-assemble into chains, and
the result is a thick gel.” (source)

Downloads:

Review of OTC Treatments for Malassezia Skin Conditions by


Tom Busby ver.3 Jan 11, 2014
MCT Oil Shower Gel & Shampoo by Tom Busby Jun 9, 2014

References:

Malassezia and Human Skin Diseases on Microbe Wiki


Malassezia furfur on Microbe Wiki
Bio lms in Dermatology
Human Infections Due to Malassezia spp.
The Rosacea Forum
Exfoliation of malassezia bio lms with Aquaphor
Candida Bio lms are stopped with Farnesol
Tinea Versicolor caused by too alkaline skin – Elle Beauty
Skin Surface pH: A Protective Acid Mantle
Skin Barrier Function

Lactic Acid excreted in sweat…get full reference….


 http://ebm.sagepub.com/content/29/8/1021.extract

July 8th, 2014 | Health


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Andrei Volokitin • 2 years ago


amazing
40 △ ▽ • Reply • Share ›

Marco Buquicchio • 3 years ago


One of the best articles on dermatitis I've ever read!
6△ ▽ • Reply • Share ›

Jared Mapes > Marco Buquicchio • 2 years ago


By FAR, the most informative and well written
articles I have read to help with my condition.
Oh and, I have ready MANY. This is superior to
all.
1△ ▽ • Reply • Share ›

Natashacbk > Marco Buquicchio • 2 years ago


I agree. It's an excellent article.
△ ▽ • Reply • Share ›

Noelle • a year ago


I stumbled onto this site after searching for information
on malassezia folliculitus (of which, this page is the
most comprehensive of those I've read) and it seems I
have found a jewel among the internet. I am loving
your other articles; thank you for sharing them. :)
2△ ▽ • Reply • Share ›

Lynley Erin Rice • a year ago


Great article, thanks :) Are you able to tell me if shea
butter and cocoa butter are within the no go range in
butter and cocoa butter are within the no go range in
the carbon chain length? Not to keen on going back to
petroleum based products after all these years, but
obviously may need to if I'm feeding the problem with
natural ones?....
1△ ▽ • Reply • Share ›

Rosalynn > Lynley Erin Rice • 9 months ago


Hi Lynley,
This is a bit of a late response but for you and
any future readers, shea butter and cocoa
butter are no goes for malassezia. Both shea
and cocoa butter contain, among other things,
stearic acid and oleic acid which are in the bad
carbon length range.

It can difficult to figure out which ones should


be avoided, but if you're looking for a way to
easily check any ingredients, I created a web
app called Sezia that will check your
ingredients against a database of ingredients
known to promote malassezia. It can be found
here. --> https://www.sezia.co/. Good luck!
△ ▽ • Reply • Share ›

Lynley Erin Rice > Rosalynn


• 9 months ago
Thank you so much!! :) Appreciate this
greatly x
△ ▽ • Reply • Share ›

Rosalynn > Rosalynn • 9 months ago


Also MCT oil is a great natural oil that
doesn't promote malassezia!
△ ▽ • Reply • Share ›

Mark • 3 years ago


When using Xylitol for acne do you eat it or use it
topically?
1△ ▽ • Reply • Share ›

mhikl > Mark • 3 years ago


Mark, this is the first article I have been able to
find on Xylitol and eczema or any skin
problems. This week I decided to try Xylitol on
my own (not finding anything about it regarding
eczema anywhere) and it has been amazing. I
do not know if it would also work for acne, but it
does for sure work for sinusitis and now it
appears it works on my eczema.
Xylitol dissolves phlegm, and therefore cleans
passages. It may do the same on your skin. I
am now ten years completely free from
sinusitis, colds and flues as I spray my nasal
passages, inner ears, eyes, eyebrows (killed
itch immediately where nothing else ever
worked—eyebrow itch attacks one in their later
years). For head orifices: 5 TBS Xylitol + 1 C
water; warm to dissolve. I use it from a Dollar
Store 250ml (1 cup) spray bottle. Use daily, or
see more

2△ ▽ • Reply • Share ›

Guest > mhikl • 2 years ago


Xylitol is not benign however. It messed
up the gut microbiome and is strongly
associated with developing kidney
stones and other oxalate issues.

https://www.ncbi.nlm.nih.go...

"The administration of fructose, xylitol or


sorbitol was associated with
the greatest renal deposition of calcium
oxalate, and glucose was
associated with by far the least."
△ ▽ • Reply • Share ›

gsaas > mhikl • 2 years ago


Mhikil,
Can you give an update on your
experience using Xylitol topically for
AD?
△ ▽ • Reply • Share ›

Guest > gsaas • 2 years ago


See above...
△ ▽ • Reply • Share ›

John Sorensen > mhikl • 3 years ago


Excellent article my friend. Great work.
△ ▽ • Reply • Share ›

Sarah Clark • a year ago


What would you recommend for SD on the scalp?
△ ▽ • Reply • Share ›

Dana Tavenner • a year ago


I think a Zpak (antibiotic) messed up my system. Doc
( ) y y
says," No'. What gets me is how can my dermatologist
not know how to make this go away when skin
disorders is his specialty?
△ ▽ • Reply • Share ›

Ismael • a year ago


Thank you so much for this gold mine of research!
Have you ever explored the application of 2.5 pH
hypochlorous acid on malessezia death? I ask
because I have a water electrolysis unit in my home
and I can produce water at any pH level as well as
hypocholrous acid (with the aid of salt). Studies show
that it has killed staph, e. coli, AIDS, and more after 30
Seconds. That being said I guess I should focus on
breaking down the bio film first... maybe a pH 11.5 to
break down the oily biofilm and then attack (spray) the
scalp with 2.5 hypochlorous acid?
https://www.ncbi.nlm.nih.go...
△ ▽ • Reply • Share ›

Ben • a year ago


Can you eleborate as to the ratio of ingredients that
you list above to maker into a topical lotion? Do you
mix all of those products together? Would really love to
know as have ordered them and desperately would
like to make it. Thanks!
△ ▽ • Reply • Share ›

Ben • a year ago


This is a great article, very, very insightful.
△ ▽ • Reply • Share ›

Dariela Myers • 2 years ago


Is there a specific ratio you would use for the
ingredients of the suggested face lotion to get it under
control?
△ ▽ • Reply • Share ›

Elliot Archuleta • 2 years ago


I'm fairly confused with this article. I've suffered with
this problem for ten years now. I am 25. I took
accutane three times (awful, I know) and I'm convinced
that exacerbated the problem. I've done my own
research, reading hundreds of articles like this and
forums, trying to find a common denominator. Since
nothing any dermatologist has prescribed in the last
decade has worked, I'm thinking the only way to at
least keep this semi contained is to change my diet. I
truly don't really have any idea what I'm doing. From
t u y do t ea y a e a y dea at do g o
what I've read, cutting out all processed foods, and
refined sugars is what seems to help people. I need to
fully understand malassezia to understand what feeds
it and what will starve it. I know nothing about pH
levels, but this article is saying to keep your pH level
between 1-4, and from what little I've read about pH
levels, that's not good for your longterm health. Not
only that, but it goes against everything I've read
regarding food consumption. I'm truly at a loss here,
and am wondering if anyone can give me insight to
this. I went on a diet last year for a couple months and
I feel like my skin cleared up pretty well. I ate mostly
fruits and vegetables, cheated rarely, and noticed a
difference. But my skin got significantly worse when
autumn came around last year and has been bad ever
since. I started doing the diet again but I haven't seen
any changes. Would love some feedback.
△ ▽ • Reply • Share ›

jenna > Elliot Archuleta • 2 years ago


You seem to have some for your information
confused. I'm going to try and help you
however be warned I'm terrible at explaining,
also i'd love to tell you about what has helped
me in my journey with acne. The article does
not state that you need to keep you pH around
1-4, the article states that your SKIN loves a pH
of around 4. A pH of 1-3 is much too acidic and
would probably burn your skin. A pH of 4 is
ideal because it’s the pH that the good bacteria
on your skin loves and will help to get rid of the
bad bacteria or fungus's etc. A lot of face
washes companies on the market know this
however there still are face washes on the
market that have a higher ph. I have a link to a
list of popular face washes and there pH

http://www.dianayvonne.com/...

see more

△ ▽ • Reply • Share ›

Julia • 2 years ago


Thank you for an informed article - I was recently
diagnosed with SD but I think I have had it for a very
long time, with flaky skin between eyebrows and upper
lip and I just thought it was dry skin as it did not bother
me too much. But last year this condition just went
mad with red inflamed and flaky skin above and
between eyebrows stretching over the sides of the
between eyebrows, stretching over the sides of the
nose and upper lip include top of they eyelids - not a
great look. So I started off with the GP's diagnosis and
they gave me Daktacort which helped a bit but the
inflammation came back so started using
hydrocortisone creams which worked but as soon as I
stopped the condition flared up again, now looking at
alternatives and recently came across a website
"Skindrone" that also talked about not applying topical
oils to the skin as this creates a feeding ground. Over
the last 5 weeks I have not been applying any oils to
my skin other than an Aveeno moisturiser which has
only liquid petroleum and dimenthicone and emolients
and using the salt bath routine. All was going really
well so decided to start using my oils again, major
disaster within days I had a flare up - so it looks like
that plant oils with their fatty acid profile is a no go area
for me now.

This is very depressing (I have my own skincare


range) that I cannot use my own creams as they
contain plant oils. I use Niacinamide (Vit B3) in my day
cream - it is an excellent ingredient (reduces redness,
increases hydration and also forms a barrier on the
skin) however, for Vitamin B3 to be stable it needs to
be in a PH of 5.5 to 6, if the ph drops as suggested in
the ariticle the Niacinamide will convert to Niacin which
can result it redness and inflammation on the skin. So I
would like to know from the author where their
reference came from in relation to using Niacinamide
at a PH 3.5 - 4.

I am about to make a cream using some of the


suggested ingredients above, using Mineral Oils in
place of plants oils, and I also like the idea of using
Xyitol too - would anyone know how much I could use
% in a cream.
Thanks
△ ▽ • Reply • Share ›

Re Gsah • 2 years ago


Great article. But I couldn't understand the
psychological part very well. Is it saying that a person
with seborrheic dermatitis should learn to contain their
emotions and to nurture agression?
△ ▽ • Reply • Share ›

GestaltReality Mod > Re Gsah • 2 years ago

Yes, that is just my hypothesis. Further


research would need to be conducted, and the
activation of individual specific muscles and
activation of individual specific muscles and
fascia would likely be different from person to
person. A sensi-motor/psychological protocol
would thus need to be individualized.
△ ▽ • Reply • Share ›

Karim Naufal • 2 years ago


Thank you from the bottom of my heart for this
extremely enlightening info. I have search the web for
year to understand my condition, facial seborrheic
dermatitis, that appeared at age 20 when I was in
emotional distress living alone for the first time for my
university studies. Everything you wrote makes so
much sense and gives me hope again.
△ ▽ • Reply • Share ›

Annie • 2 years ago


Do they mean xylitol the sweetener? If so how do you
use that on your face?
△ ▽ • Reply • Share ›

GestaltReality Mod > Annie • 2 years ago

Yes the sweeter. Mix it with slightly acetic water


and apply.
1△ ▽ • Reply • Share ›

Guest > GestaltReality • 2 years ago


Are you aware that xylitol increases
kid t f ti ?

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