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PRELIMINARY
A. Background
Cases with gastritis is one of the most common types of cases suffered by
adolescents, especially the disease is increasing among students. caused by a variety of
factors such as irregular eating patterns, wrong lifestyle and increased activity (lecture
duties) so that the student did not have time to set his diet and lazy to eat (Fahrur,
2009).
The causes of gastritis by Herlan in 2001 were excessive alcohol intake (20%),
smoking (5%), spiced food (15%), drugs (18%) and radiation therapy (2%), whereas
according to Hasna and Hurih year 2009 gastritis can also be caused by, bacterial
infections, stress, autoimmune disease, radiation and Chron's Disease.
If this gastritis disease continues to be left, will result in more severe and ultimately
stomach acid will create injuries (ulcers), known as gastric ulcers. It can even be
accompanied by blood vomiting (Arifianto, 2009). According to research Surya and
Marshall in 2007 to 2008 say gastritis that is not handled properly will lead to
complications that lead to severity. Namely cancer of the stomach and peptic ulcer.
B. Problem Formulation
1. What is gastritis?
C. Writing purpose
5. To know the treatment that can be done for people with gastritis
LITERATURE REVIEW
A. Anatomy of Physiology
Understanding
The stomach is part of the channel that can inflate mostly in the epigaster region, the
stomach consists of the top of the uterine fundus associated with the esophagus
through the pyloric orifice, located below the diagfragma in front of the pancreas and
the spleen, attached to the left of the uterine fundus (Anatomy of physiology for nursing
students 3.EGC edition: 171).
a. The ventricular fundus, the protruding part upwards to the left of the cardiac
osteoum and usually full of gas.
b. The ventricular corpus, as high as the cardiac osteoum, an indentation on the lower
part of the minor curvatura.
c. Antrum pylorus, tubular hull section has thick muscle forming spingter pylorus.
d. The minor curvatura, right side of the stomach, extends from the cardiac osteoum to
the pylorus.
e. The major curvatures, longer than the minor curvature, extend from the left side of
the cardiac ostoeum through the ventricular fundus to the right up to the inferior
pylorus. The ligamnetum of the gastrolinealis extends from the top of the major
curvature to the spleen.
f. Cardio oesteoum, is where the esophagus of the abdomen into the stomach. In this
section there is a pyloric orififum.
Gastric Function
a. Accommodates food, destroys and smooths food by peristaltic stomach and gastric
juice.
1. Pepsin functions, breaking egg white into amino acids (albumin and peptone).
2. Salt acid (HCL) function, acidify food, as an antiseptic and disinfectant, and create an
acid atmosphere on pepsinogen to become pepsin.
3. Renin its function, as yeast freeze milk and form casein from kasinogen (kasinogen on
milk protein).
4. Stomach lining : the amounts of a little break down fat into fatty acids that stimulate
gastric secretion.
B. Definitions
Gastritis or better known as ulcer is derived from the Greek gastro , which means
stomach / stomach and itis which means inflammation / inflammation. Gastritis is not a
single disease, but is formed from several conditions that all result in inflammation of
the stomach. Usually, the inflammation is the result of infection by the same bacteria
with bacteria that can cause ulcers in the stomach of Helicobacter pylori . But other
factors such as physical trauma and continuous use of some painkillers may also cause
gastritis .
In some cases, gastritis can lead to ulcers ( ulcers ) and may increase the risk of
gastric cancer. However, for many people, gastritis is not a serious illness and may soon
improve with treatment.
1. Acute Gastritis
Caused by digesting strong acids or alkalis that can cause the mucosa to become
gangrene or perforation. Acute gastritis is divided into two major lines :
2. Chronic Gastritis
D. Etiology
1. Bacterial infection .
Most of the world's population is infected by H. Pylori bacteria that live on the
inside of the mucous layer lining the stomach wall. Although it is not fully understood
how the bacteria can be transmitted, it is thought to occur through oral route or by
eating food or drink contaminated by this bacteria. H. pylori infection is common in
childhood and can last a lifetime if not treated. H. pylori infection is now known to be
the main cause of peptic ulcer and the most common cause of gastritis. Long-term
infection will cause inflammation to spread which then results in changes in the
protective layer of the stomach wall. One such change is atrophic gastritis, a condition
in which the acid-producing glands are slowly damaged. Researchers concluded that low
levels of stomach acid can result in toxins produced by cancer not to be destroyed or
excreted completely from the stomach, increasing the risk of stomach cancer. But most
people with chronic H. pylori infection have no cancer and no symptoms of gastritis,
indicating that there are other causes that make some people vulnerable to these
bacteria while others are not.
Alcohol can irritate and erode the mucosa in the gastric wall and make the stomach
wall more susceptible to stomach acid even under normal conditions.
4. Cocaine use .
Cocaine can damage the stomach and cause bleeding and gastritis.
5. Physical stress .
Physical stress due to major surgery, traumatic injury, burns or severe infections can
cause gastritis and ulcers and bleeding in the stomach.
6. Autoimmune disorder .
Autoimmune atrophic gastritis occurs when the immune system attacks healthy cells
that are in the wall of the stomach. This results in inflammation and gradually dilutes the
stomach wall, destroys the gastric acid producing glands and disrupts the production of
intrinsic factors (ie a substance that helps the body absorb vitamin B-12). Lack of B-12, in
the end, can lead to pernicious anemia, a serious con- sumption that if not treated can
affect the entire system in the body. Autoimmune atrophic gastritis occurs mainly in the
elderly.
7. Crohn's disease .
Although the disease usually causes chronic inflammation of the gastrointestinal wall,
it can sometimes also cause inflammation of the gastric wall. When the stomach is
affected, the symptoms of Crohn's disease (ie stomach pain and diarrhea in fluid form)
appear more prominently than symptoms of gastritis.
Bile (bile) is a fluid that helps digest fats in the body. This liquid is produced by the
liver. When released, the bile will pass through a series of small channels and headed to
the small intestine. Under normal circumstances, a ring-like sphincter muscle (pyloric
valve) will prevent bile from flowing back into the stomach. But if this valve does not
work properly, then the bile will enter the stomach and cause inflammation and
gastritis.
10. Other factors .
Gastritis is often also associated with other health concepts such as HIV / AIDS,
parasitic infections, and liver or kidney failure.
E. Pathophysiology
Acute Gastritis
Excess alcohol, eating too many foods that contain nitrates (preservatives) or too
acid (vinegar), caffeine as in tea and coffee and smoking can trigger the occurrence of
gastritis. Because these materials when too often in contact with the wall of the
stomach will trigger excessive gastric acid secretion that can erode the gastric mucosal
layer.
Then the old psychological and physiological stress can cause gastritis. Stress such
as shock, sepsis, and trauma cause gastric mucosal ischemia. Gastric mucosal ischemia
results in increased mucosal permeability resulting in H diffusion into the mucosa. The
+
mucosa can no longer withstand excess acid causing edema and then damaged.
Chronic Gastritis
Analogics
Chemical material
Alcohol
Physical stress
Damage of the gastric mucosal barrier
.
G. Clinical manifestations
a. Acute Gastritis
Dyspepsia syndrome of epigastrum pain, nausea, bloating, vomiting, is one of the most
common complaints. Also found gastrointestinal bleeding in the form of hematemesis
and melena, if done a deep lebig history, there is a history of use of drugs or certain
chemicals . ( capita selekta medical vol 1 1st edition FKUI matters: 492)
b.Gastritis Chronicles
H. Complications
1. Acute Gastritis
2. Chronic Gastritis
I. Management
Acute Gastritis
The main factor is eliminating its etiology. Gastric diet, with small portions and often.
Drugs intended to regulate gastric acid secretion, such as H receptor antagonist ₂ ,
proton pump inhibitors, anticholinergics, and antacid. Also referred to as cytoprotectors,
sucralfat and prostaglandin. . ( capita selekta medicine volume 3rd edition FKUI matters:
493)
Chronic gastritis
Eradication therapy is given for 1-2 weeks with regard to cost efficiency. The
treatment regimen divided by 3, triple, kuadrel, and dual, but Yag commonly used triple
and kuadrel. If therapy fails, use terapikuadrel. Patients are considered cured, only if
after 4 weeks of therapy is completed CLO and PA negative examination results, in
addition therapy is considered failed. Complete regimen and dose of eradication
therapy. . ( capita selekta medicine volume 3rd edition FKUI matters: 493-494)
In gastritis, its management can be done with (medical and non medical), as follows:
a. Acute Gastritis
8. If gastritis occurs due to ingestion of a strong base, use a watery orange juice or dilute
vinegar.
9. Stomach reactions are needed to overcome the obstruction of polirus.
b. Chronic Gastritis
1. Can be solved by modifying the patient's diet, the soft eating diet is given little
but more often.
2. Reduce stress
J. Investigations
a. Blood examination
This test is used to check whether there is H. pylori in the blood. A positive test results
showed that the patient had contact with bacteria at some time in his life but that did
not indicate that the patient had an infection. Blood tests can also be performed to
check for anemia that occurs due to gastric bleeding due to gastritis.
A diagnostic method based on the principle that urea is altered by H. pylori urease in
the stomach into ammonia and carbon dioxide (CO ). CO rapidly absorbed through the
2 2 is
c. Stool examination
This test checks whether there is H. pylori bacteria in the feces or not. Positive
results may indicate the occurrence of infection. Examination is also done on the
presence of blood in the feces. This shows the presence of bleeding in the stomach.
This test will see signs of gastritis or other digestive diseases. Usually will be required
to swallow the barium liquid first before X-rays. This liquid will coat the gastrointestinal
tract and will be seen more clearly when the x-rays.
f . Gastric Analysis
g. Stimulation analysis
Can be done by measuring the maximum acid expenditure (MAO, maximum acid
output) after administration of drugs that stimulate the secretion of acids such as
histamine or pentagastrin. This test is to determine whether it is aclorhidria or not.
GASTRITIS NURSING ASSISTANCE
ASSESSMENT.
Anamnese include:
1. Name : Ny. M
2. Age : 58 thn
3. Sex : Female
7. Religion : Islam
8. Level of education : for people with low / low education levels gaining knowledge
about gastritis, it will underestimate this disease, even just consider gastritis as an
ordinary stomachache and will eat foods that can cause and aggravate this disease.
a) Main complaints : Pain in the pit of the liver and lower right abdomen.
b) when the medical history is : Covering the course of the disease, the beginning of
your symptoms client, complaints arise suddenly or gradually perceived, can trigger an
effort to resolve the issue.
c) Past medical history : Includes diseases related to current illness, hospital history , and
history of drug use.
1. B1 ( breath) : takhipnea
2. B2 (blood) : tachycardia, hypotension , dysrhythmias , weak peripheral
pulse, slow peripheral fill, pale skin color.
1. Activity / Rest
2. Circulation
- skin / mucous membrane weakness , sweating (indicating shock status, acute pain,
psychological response)
3. Integrity of the ego
4. Elimination
Symptoms : previous hospital care history due to gastroenteritis (GE) bleeding or GE-
related problems, eg peptic or gastric wounds , gastritis, gastric surgery, gastric area
irradiation. Changes in the pattern of defecation / faecal characteristics.
5. Food / Liquid
Signs : vomiting with dark or bright red coffee, with or without blood clots, dry mucous
membranes, decreased mucosal production, poor skin turgor (chronic hemorrhage).
6. Neurosensi
Signs : the level of consciousness can be disturbed, range from rather likely to sleep,
disorientation / confusion, to fainting and coma (depending on the volume of circulation
/ oxygenation).
7. Pain / Comfort
Symptoms :- pain, described as sharp, shallow, burning, sore, sudden pain can be
accompanied by perforation. Sense of discomfort / distress vaguely after eating a lot
and lost by eating (acute gastritis).
- left / middle epigastric pain / or spread to the back occurring 1-2 hours after
eating and lost with antacids (gastric ulcers).
- trigger factors : food, cigarettes, alcohol, use of certain drugs (salicylate, reserpine,
antibiotics, ibuprofen), psychological stressors.
Signs : face wrinkled, careful on the areas of pain, pale, sweaty, attention narrowed.
8. Security
Symptoms : allergy to drugs / sensitive eg: ASA
Symptoms : the use of prescription drugs / sold free containing ASA, alcohol, steroids.
NSAIDs cause GI bleeding. The present complaint may be acceptable due to ( eg:
anemia) or unrelated diagnosis (eg head trauma), intestinal flu, or severe vomiting
episodes. Old health problems such as cirrhosis, alcoholism, hepatitis, eating disorders
(Doengoes, 1999, p. 455).
NURSING DIAGNOSES
2. The volume of fluid is less than the body's needs associated with inadequate intake
and excessive liquid output (nausea and vomiting)
- Pain scale 0.
- Client can relax. 4. Teach distraction and 4. Can make clients better
- The general state of recluse techniques. and forget about pain.
the client is good.
- BB increases
5. Consult with a team of 5. Accelerate the fulfillment
- The difficulty of nutritionists in the menu. of nutritional needs with the
swallowing is reduced right target menu.
COVER
A. CONCLUSION
Gastritis is not a single disease, but is formed from several conditions that all result
in inflammation of the stomach. Usually, the inflammation is the result of infection by
the same bacteria with bacteria that can cause ulcers in the stomach of Helicobacter
pylori. But other factors such as physical trauma and the continued use of some
painkillers may also cause gastritis. Although many conditions can cause gastritis, the
symptoms and signs of the disease are similar to each other.
B. CRITICISTS AND SUGGESTIONS In order to refine this paper , I really expect criticism
and suggestion from Supervisor and other group's friends.
BIBLIOGRAPHY
Doengoes , Marilyn.E.dkk.2006. Buku Ajar Ilmu Disakit Dalam . Jakarta: Center for
Publishing Department of Internal Medicine FKUI
Wilkinson, Judith M. 2007. A Nursing Diagnosis Handbook with NIC Interventions and
NOC Criteria . Jakarta: EGC