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Oblique Comminuted
Transverse Spiral Segmental
Describing Fractures
Displacement Angulation
Orthopedic Emergencies
Pediatric Fractures
Salter-Harris fractures involve the epiphysis, or cartilaginous
epiphyseal growth plate, near the ends of the long bones in
children
Named after the two physicians who devised the classification
system for naming these fractures
New bone material needed for elongation of bones during growth
is provided by specialized cells within the physis
When growth is complete, transformation of the physis into bone
occurs, ultimately fusing with the surrounding bone
Salter-Harris fractures cannot occur in adults
Pediatric Fractures
Damage to the epiphyseal plate during bone growth can destroy
all or part of its ability to produce new bone
This may result in an aborted or deformed bone growth of bone
The earlier a Salter-Harris fracture occurs, the more likely the
chance of a deformity will occur
Approximately 15% of growth plate fractures will have long term
bone growth disturbance
Fracture pattern is also a significant factor in the development of
deformity
Orthopedic Emergencies
Salter-Harris Classification
Salter-Harris Classification
Initial Treatment - Splinting
Control pain and swelling
Reduce deformity/dislocations
Immobilization of fracture, sprain, or injury
Splinting & Immobilization
Goals
– Relieve pain
– Augment healing
– Stabilize fracture
– Prevent further injury
Splinting
Splinting and immobilization of fractures is the mainstay of
emergency orthopedics
Most fractures can be immobilized with a simple splint
Fracture type will dictate the splint required to immobilize it
Most splints used in the emergency department are either OCLTM
or Ortho-GlassTM
The goal of fracture immobilization is to protect the damaged
bone, while keeping it in anatomic position; this will facilitate
healing with no anatomic defect
There are numerous types of splints; however, one can modify a
splint to immobilize almost any fracture
Splinting
Immobilization facilitates the healing process by decreasing pain
and protecting the extremity from further injury
Splinting maintains bony alignment
Splinting also reduces motion; by limiting early mobility, edema
can be reduced
Splints can be either plaster of Paris or fiberglass (OrthoGlassTM)
Plaster of Paris is the most widely used material for splinting in
the ED; however, the fiberglass splints are becoming
extremely popular, as they are easy to apply and less messy
Splinting Advantages
Over Casting
Ease of application
Short-term immobilization
Allows continued swelling to prevent
complications
Patient removal
Splinting Indications
Fractures
Deep laceration/large abrasions
Tendon lacerations
Inflammatory disorders (gout,
tenosynovitis)
Deep space infections (hand, feet, joints)
Multiple trauma
Splinting
Which type of splint do you use?
Most upper extremity injuries can be managed using a long arm
posterior splint
Sugar tong, ulnar gutter and thumb spica are also used
Finger injuries can be managed with foam finger splints or hard
plastic splints
Shoulder injuries can be managed with a sling/swathe, or
shoulder immobilizer
Lower extremity injuries can be managed with a knee
immobilizer or posterior mold splint; ankle injuries can be
managed with preformed splints or posterior mold splint
Principles of Splinting
Assess ABC's and treat life-threatening
situations first
Identify/assess neurovascular structures at
risk
Early orthopedic consultation for open
fractures or fracture-dislocations
Select appropriate immobilization technique
Document and dress open wounds
Principles of Splinting (cont’d)
Remove all clothing and constrictive devices
from extremity (jewelry, rings)
Align severely angulated fracture
Protect bony prominence
Assess neurovascular status immediately
before and after splinting
If periodic wound care is required, consider
a removable splint
Complications
Ischemia
Plaster burn
Pressure sores
Infection
Dermatitis
Joint stiffness
Discharge Instructions
Elevation
Ice bags/cold packs
Allow setting of splint
Avoid getting splint wet
Clear follow-up instructions
Check for signs of vascular insufficiency
Risk Management Issues
Always document neurovascular status before and
after splint application
Always document neurovascular status before
and after any fracture or joint reduction
Remove all rings on hands/toes before splint
application
Clearly document follow-up instructions:
– with whom
– when to see orthopedic physician
– when to return to the emergency department
Fracture Pathophysiology
Inflammatory Phase
After the initial fracture, microvessels that cross the fracture line
are transected; this results in ischemia to the damaged bone
ends
Damaged bone ends necrose, which triggers an inflammatory
response
Inflammatory phase is brief, but creates the tissue environment
for the reparative phase
Fracture Pathophysiology
Reparative Phase
The reparative phase begins with granulation tissue infiltrating
the fracture area
Granulation tissue contains cells that secrete and form collagen,
cartilage and bone; these form the callus, which eventually
surrounds the fractured ends of the bone
Callus is responsible for stabilizing the fractured bone ends
As the fracture heals, the callus becomes mineralized and very
dense
The necrotic edges of the fracture fragments are attacked by
osteoclasts, which resorb bone
Fracture Pathophysiology
Remodeling Phase
Remodeling is the final phase of bone healing
The bone gradually regains its original shape, contour and
strength
Remodeling often lasts years
Callus is resorbed, new bone laid down by osteoblasts
The trabeculae, linear densities easily seen on normal bone, are
the end result of the physiologic process that remodels bone
and provides maximum strength in relation to the amount of
bone used
Orthopedic Emergencies
Disproportionate pain
Weakness
Pain with passive stretching of compartment
muscles
Hypesthesia or paresthesias of nerves
within the compartment
Normal pulses and capillary refill
Compartment Syndromes
Findings (the 6 P’s)
Pain Parasthesias
Pallor Paralysis
Pulselessness Poikilothermic
Compartment Syndromes
Diagnosis
Elevated compartment pressures
– Normal 0-8 mmHg
– Damage begins with pressures of 30-45
mmHg
– Measured with a catheter inserted into
the individual compartment in question
Compartment Syndrome Treatment
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