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Rajesh Shetty, Arpita Agarwalla, Pritesh Jagtap, Nikita Patel, Ankush Katkade, Shrutika Somani
Correspondence to: Dr. Arpita Agarwalla, PG Student in Department of Conservative Dentistry and Endodontics, Dr. Dnyandeo Yashwantrao
Patil Dental College and Hospital, Pimpri, Pune - 411 018, Maharashtra, India. E-mail: arpita.agarwal19@gmail.com
2013 with the following Medical Subject Headings (MeSH) The diagnosis can be difficult to establish because the
terms and/or key words in various combinations: Cracked fracture can mimic other conditions such as sinusitis,
tooth, vertical root fracture, tooth fracture, split tooth, cuspal atypical facial pain, etc., Treatment is usually extraction
fracture odontalgia, bite tests, and splinting. About 272 articles of the tooth.[13]
were found, out of which many were excluded based on the
exclusion criteria mentioned above and 60 of the articles were Etiology
used for this review. The etiology of CTS is multifactorial. Guersten et al.,[14] stated
that “excessive forces applied to a healthy tooth or physiologic
Discussion forces applied to a weakened tooth can cause an incomplete
fracture of enamel or dentine.”
Epidemiology
The presence of a cracked tooth occurs primarily in adulthood. Lynch et al.,[15] have subdivided the causes of cracks into four
Contradictory data were reported regarding the correlation major categories: Restorative procedures, occlusal factors,
between a patient’s age and the occurrence of CTS. developmental conditions, and miscellaneous factors.
grooves, or a bifurcation. Thus, maxillary premolars Successful treatment and favorable prognosis would depend
are significantly more susceptible to fracture than on an accurate and early diagnosis.
mandibular premolars. Additionally, an extensive pulp
space, a “steep cusp/deep groove” inter‑relationship A detailed history is imperative along with a thorough
between the maxillary and mandibular premolars, assessment of all symptoms, though these vary with the depth
and the resulting wedging effect of the prominent and orientation of the crack.
facial cusps of mandibular premolars, contribute to
If the crack is directly visible, diagnosis can be fairly simple.[5]
the increased susceptibility to fracture of maxillary
This is possible in cases where there is exogenic staining from
premolars.[24,25]
food or beverages. But more often than not, common mesiodistal
• Miscellaneous factors‑Miscellaneous factors include
cracks are not visible because most of these teeth present with an
wasting diseases like abrasion, erosion or attrition.
overlying occlusal or proximal restoration. Hence removal of these
acidogenic extensive loss of enamel and dentin,
restorations may prove to be useful.[28] With larger restorations,
andbulimia or anorexia nervosa, may also increase the
cracks tend to be more superficial and thereby produce fewer
risk of a fracture.[14]
symptoms, whereas with smaller restorations cracks tend to be
In recent years, the lingual barbell or tongue piercing has been deeper and closer to the pulp.[31] Initial cracks may not be visible
identified as a frequent cause for cracks on teeth.[26] because they are too small to be detected by the naked eye.[32]
With age, dental tissues become more brittle and less elastic, Many authors have suggested the use of stains like gentian
thus even forces which were otherwise within normal violet or methylene blue to stain the fracture line and make it
physiologic limits may exceed the elastic limits of an aged visible.[30] But this technique still has its disadvantages-first,
dentition, making it more susceptible to fracture.[27] that staining may take many days to become effective,
hence requiring an interim restoration[5] which may further
Signs and symptoms compromise the structural integrity of the tooth. Furthermore,
The most classic symptom of CTS is a history of sharp pain when after the use of these dyes, placing a definitive aesthetic
biting or while consumption of hot/cold beverages.[28] This pain on restoration may prove to be difficult.[28]
biting increases as the applied occlusal force is raised.[27] The pain
usually occurs on release of pressure when fibrous foods are eaten, Magnifying loupes and transillumination using a fiber‑optic
hence it is known as “rebound pain.”[12] The alternative stretching device have been considered instrumental in the diagnosis of
and compressing of the odontoblastic processes located in the a cracked tooth.[16]
crack has been thought to be the cause for this short, sharp pain.[24] One of the classic symptoms of CTS, which most dental
This pain has also been accounted to the sudden movement operators consider to be an important indicator, is cold
of fluid present in dentinal tubules which occurs when the sensitivity and sharp pain on biting hard or tough food which
fractured portions of the tooth move independently of one ceases on the release of pressure.
another, which results in the activation of myelinated A‑type
This “relief pain” can be replicated by bite tests.[15] To
fibers within the dental pulp. Hypersensitivity to cold may
accurately diagnose an incomplete fracture by the use of any
also occur as a result of the seepage of noxious irritants
of the following-orange wood sticks, cotton-wool rolls, rubber
through the crack, which results in the subsequent release of
abrasive wheels such as a Burlew wheel or the head of a number
neuropeptides causing a concomitant lowering in the pain
10 round bur in a handle of a cellophane tape.
threshold of unmyelinated C‑type fibres within the dental
pulp.[29] In order to localize the cusp affected by CTS, a wood stick can
be rested on each individual cusp of the suspected tooth and
Very often fractures are not diagnosed in time because of
the patient asked to bite. According to Kruger, pain produced
which they may enter the pulp chamber, causing pulpal
by release of pressure confirms a case of CTS.[33]
inflammation and necrosis. Fractures extending to the root
generally cause periodontal inflammation. Thus, a localized The use of commercially available diagnostic tools like
periodontal breakdown adjacent to a restored tooth frequently Fractfinder (Denbur, Oak Brook, IL, USA) and Tooth Slooth
indicates a fracture.[5] II (Professional Results Inc., Laguna Niguel, California,
USA) have found to be more sensitive and accurate in the
Diagnosis identification of the affected/involved cusp when compared
Diagnosing CTS has proved to be difficult even for the with the more conventional tools.[1]
most experienced dental practitioners, the reason being that
associated symptoms are usually very variable and sometimes Most cracks propagate in a mesiodistal direction, because of
even bizarre.[30] which radiographs are barely of any use.[15] However in the
rare cases where the fracture occurs bucco‑lingually they are Definitive therapy
valuable in the diagnosis.[34] In case of non splintering teeth, intra coronal restorations,
without cuspal coverage may also be used. Dental amalgam,
But Cone Bean Computed Tomography (CBCT) may prove to
composite resin and glass‑ionomer cements are most commonly
be useful in many cases.
used. The strength of the tooth can be restored by using these
materials.[38]
Treatment
Immediate therapy
Amalgam overlays have been found to increase the fracture
To avoid irreversible damage, it is imperative that a cracked
energies of cracked teeth to levels equivalent to that of intact
tooth be treated as soon as possible.
teeth.[39] Hence direct restorations with cuspal coverage have
Occlusal adjustment of affected teeth must be done immediately been advocated as a primary mode of treatment.[31]
to reduce the stress on the tooth and prevent further damage
to the tooth. In cases where aesthetics is not a matter of great concern,
cast metal inlays with cusp coverage or partial crowns with
If the tooth in question presented with a preexisting restoration, it circumferential external splinting are applied or else, adhesive
should be removed. This may cause the affected cusp to “splinter ceramic restorations can also be used.
off” and the further treatment protocol can then be decided.[35]
Full coverage crowns are the most appropriate form of
In case there is no splintering, immediate immobilization
restoration for cases where the crack extends from the occlusal
should be employed, using an “immediate extra‑coronal
incline to the cervical third of the clinical crown.[36]
circumferential splint.” A copper ring or a stainless steel
orthodontic band can be used for this purpose.[5] It has been reported by a number of authors that the loss of
vitality following the application of full coverage single unit
The use of full coverage acrylic provisional crowns has been
crowns is in the range of 15-19%. Loss of pulpal vitality is an
advocated for “immediate splinting.”[36]
obvious problem following the preparation of teeth to receive a
Another available option is the use of bonded composite resin full coverage crown. In CTS, reversible pulpitis would already
to splint the teeth, which is known as a “direct composite be a pre‑ existing condition so the problem appears to be further
splint” (DCS).[37] compounded [Figure 1].[15,40,41]
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Figure 1: Treatment options
26. DiAngelis AJ. The lingual barbell: A new etiology for the cracked‑tooth 38. Geurtsen W, Garcia‑Godov F. Bonded restorations for the prevention and
syndrome. J Am Dent Assoc 1997;128:1438‑9. treatment of the cracked tooth syndrome. Am J Dent 1999;12:266‑70.
27. Signore A, Benedicenti S, Covani U, Ravera G. A 4 to 6 year retrospective 39. Hodd JA. Biomechanics of the intact, prepared and restored tooth; some
clinical study of cracked teeth restored with bonded indirect resin clinical implications. Int Dent J 1991;41:25‑32.
composite onlays. Int J Prosthodont 2007;20:609‑16. 40. Saunders WP, Saunders EM. Prevalence of periradicular periodontitis
28. Banerji S, Mehta SB, Millar BJ. Cracked tooth syndrome. Part 1: Aetiology associated with crowned teeth in an adult Scottish subpopulation. Br Dent
and diagnosis. Br Dent J 2010;208:459‑63. J 1988;185:137‑40.
29. Davis R, Overton JD. Efficacy of bonded and nonbonded amalgams 41. Cheung GS, Lia SC, Ng RP. Fate of vital pulps beneath a metal ceramic
in the treatment of teeth with incomplete fractures. J Am Dent Assoc crown or a bridge retainer. Int Endod J 2005;38:521‑30.
2000;131:496‑78. 42. Qian, Yunzhu, Zhou, Xuefeng, Yang, Jianxin. Correlation between cuspal
30. Liu HH, Sidhu SK. Cracked teeth‑treatment rational and case inclination and tooth cracked syndrome: A three‑dimensional reconstruction
management: Case reports. Quintessence Int 1995;26:485‑92. measurement and finite element analysis. Dent Traumatol 2013;3:226‑33.
31. Homewood CI. Cracked tooth syndrome‑incidence, clinical findings and 43. Clark LL, Caughman WF. Restorative treatment for the cracked tooth.
treatment. Aust Dent J 1998;43:217‑22. Oper Dent 1984;9:136‑42.
32. Caulfield JB. Hairline tooth fracture: A clinical case report. J Am Dent 44. Tan L, Chen NN, Poon CY, Wong HB. Survival of root filled cracked teeth
Assoc 1981;102:501‑2. in a tertiary institution. Int Endod J 2006;39:886‑9.
33. Kruger BF. Cracked tooth syndrome. Letter to the editor. Aust Dent J 45. Nakajima Y, Shimada Y, Miyashin M, Takagi Y, Tagami J, Sumi Y.
1984;29:55. Noninvasive cross‑sectional imaging of incomplete crown fractures (cracks)
34. Turp JC, Gobetti JP. The cracked tooth syndrome: An elusive diagnosis. using swept‑source optical coherence tomography. Int Endod J 2012;45:933‑41.
J Am Dent Assoc 1996;127:1502‑7.
35. Fox K, Youngson CC. Diagnosis and treatment of the cracked tooth. Prim
How to cite this article: Shetty R, Agarwalla A, Jagtap P, Patel N,
Dent Care 1997;4:109‑13.
Katkade A, Somani S. Cracked tooth syndrome. J Dent Res Sci Develop
36. Gutherie RC, Difiore PM. Treating the cracked tooth with a full crown. 2014;1:51-6.
J Am Dent Assoc 1991;122:71‑3.
37. Banerji S, Mehta SB, Millar BJ. Br Dent J 2010;208:459-63. Source of Support: Nil Conflict of Interest: No conflict of interest.
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