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no motile capsule-like
Bacteria Crecimiento general pathogenesis hemolisis catalase virulence diagnosis other
caracteristics factors
structure
PCR
Bacteria Crecimiento general pathogenesis hemolisis catalase virulence diagnosis other
caracteristics factors
cause
endogenous
infection
grastroenteritis
(exogenous)
Spread by fecal-oral
core trans.
polysaccharide
lipid A
Produce H2S
flagella)
facultative anaerobes
Colonizes: skin,
URtrack and
colon
enf. nosocomiales
Bacillus anthrasis
serpentine chains Poly-D-glutamic acid plasmid pX01 Malachite green bioterrorism agent
capcid (Px02 stain (spores)
non hemolitic plasmid) edema factor(EF, aquired via
inherited adenylate DFA capsula inhalation,
nonmotile spores cyclase) polypeptide inoculation and
ingestion
Protective blood agar: crece cid
antigen(PA,binding medussa head and si no tiene la capsula
domain) ground glass y las 3 toxinas no es
appearance (relieve) patogeno!!
Lethal factor(LF,
stimulates release of
Lysis with gamma
TNF-gamma, IL-1B,
phage(esp.)
proinflamatory factors) requieres thiamine to
grow
penincillin(susceptibl
e, string of pearls
formation)
cereolysin: hemolisis
Phospholipase C:
lethinase
spores
Clostridium perfinges
no motile(unico lethinase alpha: hemolysis gram stain part of the normal
clostridium) (phospholipid C) flora
Beta: necrotizing enterotoxins on feces
double zone Protease type A is responsible
hemolisis epsilon: increase ana blood agar for most human
vascular permeability
Bacteria physiology and virulence toxins diagnosis other
structure/ gen charac.
Clostridium tetani spores toxins cant be aislated on spores found in soils and
motile oxygen labile lab, diagnosis is based can colonise GI tract
emolysin on symptoms
terminal spores (tetanolysin)
beta:
deoxyribonuclease
gamma:
hyalouronidase
delta: hemolysin
neuroaminidase: alters
cell membrane