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*Pulse oximetry is
used in
measuring
7. Assessed oxygen
urine output. saturation. The
normal oxygen
saturation should
be maintained at
90% or higher. As
shock progresses,
aerobic
metabolism stops
and lactic acidosis
occurs, resulting
in the increased
DEPENDENT: level of carbon
dioxide and
1. decreasing pH.
Administered
supplemental
oxygen at 3 *The renal system
LPM, as compensates for
needed. low BP by
retaining water.
2. Measured Oliguria is a
CO and other classic sign of
functional inadequate renal
parameters as perfusion from
appropriate. reduced cardiac
output.
*Increases the
amount of oxygen
available.
*Cardiac index,
preload and
afterload,
contractility and
cardiac work can
be measured
noninvasively.
6. Proper body
6. Reviewed
mechanics reduces
proper body
the risk of muscle
mechanics or
strain, injury, or
techniques for
pain. It also
participation in
increases
activities
8. Prevents
contractures/
footdrop and
facilitated use
when/ if function
8. Positioned in
returns. Flaccid
prone position
paralysis may
once or twice a
interfere with
day if client can
ability to support
tolerate.
head, whereas
spastic paralysis
may lead to
deviation of head
to one side.
9. Pressure points
over bony
prominences are
most at risk for
9. Inspected decreased
skin regularly, perfusion/
particularly ischemia.
over bony
prominences.
Gently massage
10. Minimizes
any reddened
muscle atrophy,
areas.
promotes
circulation, and
10. Exercise
helps prevent
therapy: Begun
passive ROM to contractures.
all extremities
on admission.
Encourage
exercises such
as
11. Involved the
quadriceps/glut
client and SO in
eal exercise,
setting for
squeezing
increasing
rubber ball.
participation in
activities/ exercise.
11. Involved the
client and SO in
setting for
12. Individualized
increasing
program can be
participation in
developed to meet
activities/
particular needs/
exercise.
deal with deficits
in balance,
Collaborative:
coordination,
12. Consulted strength.
with physical
therapist
regarding
active, resistive
exercises and
client
ambulation.
CUES NURSING SCIENTIFIC GOAL INTERVENTIO RATIONALE EVALUATI
DIAGNOSIS RATIONALE N PLAN ON
Subjective:Deficient Fluid In hypovolemic Short term:
Volume related shock, there is
Oo After 1-2 Independent: Goal
to hemorrhage inadequate
nagmamata weeks of partially
from ruptured circulating blood 1. Obtained 1. Helps estimate
hiya pero nursing met.
esophageal volume as a result history from total depletion.
maluyahon, interventions,
varices from of an advanced client and Symptoms may
as the client will
portal decline in the significant have been
verbalized be able to
hypertension cardiac output. The other (S0) present for
by the SO. demonstrate
as evidenced vasoconstrictive related to varying amounts
adequate
by upper compensatory duration and of time hours
hydration as
gastrointestina mechanisms of the intensity of to days.
evidence by
l bleeding. body have failed at symptoms Presenvce of
stable vital
this time, hence the such as infection process
microcirculation signs, bleeding results in fever
dilates, leading to palpable and and
further blood loss. peripheral vomiting hypermetabolic
pulses, good and state, increasing
skin turgor excessive insensible fluid
and capillary urination. losses.
refill, 2. Hypovolemia
individually may be
appropriate manifested by
Objective:
urinary hypotension and
*Decreased
output, and tachycardia.
hemoglobin: 2. Monitored
electrolyte Estimates of
91 g/ml vital sign,
levels within severity of
(reference: specifically
normal range. hypovolemia
120- BP and the
may be made
160g/ml) RR.
when clients
*Decreased
systolic BP drops
hematocrit:
more than 10
0.29
mmhg from a
(reference:
recumbent to
0.40-0.54)
sitting or
*Body
standing
malaise
position.
*Body
Increased work
weakness
of breathing
*Pallor
shallow , rapid
*Cold
respirations and
clammy
presence of
skin
cyanosis may
indicate
respiratory
fatigue and
client is losing
ability to
compensate for
acidosis.
3. Indicators of
level of
hydration and
3. Assessed adequacy of
peripheral circulating
pulses, volume.
capillary
refill, skin
4. Provides
turgor, and
ongoing
mucous
estimate of
membranes.
volume
replacement
4. Monitored needs, kidney
fluid intake function, and
and output effective ness of
(I&O); note therapy.
urine
specific
5. Provides the
gravity.
best assessment
of current fluid
status and
adequacy of
fluid
5. Weighed
replacement.
patient daily.
6. Changes in
mentation can
be due
electrolyte
abnormalities,
6. Investigated acidosis,
changes in decreased
mentation cerebral
and perfusion, or
sensorium. developing
hypoxia,
Regardless of
the cause ,
impaired
consciousness
can predispose
client to
aspiration.
Dependent:
Administered IV
fluids, as
indicated.
CUES NURSING SCIENTIFIC GOAL INTERVENTIO RATIONALE EVALUATI
DIAGNOSIS RATIONALE N PLAN ON
Short term: Independent
SUBJECTIV Impaired Gas Dyspnea typically Goal
E Exchange occur during shock Partially
After 2 hours 1. Assessed 1. Manifestation of
related to because of Met.
Baga man of nursing respiratory respiratory
decreased decreased tissue
iton hiya intervention rate, depth, distress are
tissue perfusion. The and ease. dependent on
hin the client will
perfusion as a respiratory rate and indicative of
nagkukuri be able:
result of the increases as the the degree of
pagginhaw
decrease in oxygen-carrying lung
a han
cardiac output capacity of the involvement and
amon a) Demonstr
brought about blood decreased. underlying
pagdad-a ate
by general health
ngadi. There is imbalance improved
hypovolemia. status.
Gintauran in amount of oxygen ventilatio
nla hiya demand and supply n and
intawon due to decreased oxygenati 2. Observed 2. Cyanosis of
on of color of skin,
hin amount of oxygen in nailbeds may
tissues by mucous
oxygen, the blood due to represent
ABGs membranes
as decrease cardiac vasoconstriction
within and nail bed,
or the bodys
verbalized output caused by noting
clients response to
by fluid loss that had acceptabl presence of fever or chills;
patients caused the e range peripheral however,
daughter. hypovolemic shock and cyanosis or cyanosis of
where the body cant absence central earlobes,
compensate the loss of cyanosis. mucous
of blood. symptom membranes ,
s of and skin around
(Focus on respirator the mouth is
Pathophysiology by y distress. indicative of
Objective: Barbara L. Bullock b) Participat systemic.
Dizzines and Reet L. Henze) e in
s actions to
Weakne maximize
oxygenati 3. Tachycardia is
ss 3. Monitored usually present
on.
BP: 70 heart rate as a result of
palpato and rhythm. fever and
ry dehydration, but
PR: 116 may represent a
BPM response of
RR: 24 hypoxia.
CPM
4. These measures
promote
maximal
4. Elevated
inspiration and
head and
enhance
positioned at
expectoration of
MHBR.
secretion to
Encouraged
improve
deep
ventilation.
breathing
and effective
coughing. 5. Identifies
problems, such
as ventilator
Collaborative
failure; follows
5. Monitored progress of
ABGs and disease process
pulse or improvement.
oximetry.
6. Increase
amount of
oxygen available
for myocardial
6. Administered uptake;
oxygen as Oximetry
ordered; measures
maintained peripheral
continuous oxygen-
oximetry. saturation.
7. With increasing
hypoxia,
mechanical
ventilation may
be necessary to
7. Prepared for oxygenate the
intubation client
and adequately.
mechanical
ventilation if
hypoxia
increases.
(Nursing Care
Plans-
Guidelines for
individualizing
Client Care
Across the Life
Span by Marilyn
e.
Doenges,Mary
Frances
Moorhouse,
Alice C. Murr)