Cell cycles consist of initial growth phase(G1),DNA replication(S), a gap phase(2)

and mitosis(M),after the cell may enter the resting state.(G0)

The cycle is driven by a number of positive and negative regulatory
phosphorylation and dephosphorylating events involving protein kinases, protein
phosphates,cyclins,cyclin-dependant kinases and cyclin-dependant kinases
inhibitors.
Unreplicated /damaged DNA blocks the progression of the cell cycle at
checkpoints, including the late (G1) and late (G2.)
During replicative cycles of virus, many types of abnormal replicative cycle do
occur. They are as follows:
Incomplete viruses: defect during assembly of viral components, some of the
daughter virions that are produced may not be effective. Example of such
incomplete virus is the influenza virus that shows a high hemagglurination titer
but with a low infectivity, this is known as von magnum phenomenon.
Pseudovirions are the virus that occasionally enclose host cell nucleic acid
instead of viral nucleic acid. These therefore, are non-infective and lack the
capability to replicate.
Abortive infections: in this type of infection, the virus components, may be
synthesized but the maturation is defective. The progeny virions are either not
released or are non-infectious. This type of infection occurs due to infection of
wrong host cell by the virus.
Mutation (point mutation): some virus have a genome made from RNA, instead of
DNA. The enzymes that copy RNA genomes are more prone to making errors
than are the enzymes that copy DNA genomes; RNA-based viruses therefore
have a higher rate of mutation. This is why its generally harder to develop
vaccines against RNA-based viruses than against DNA-based virus- they change
too fast! Some viruses, e.g. the flu, have a genome thats in multiple parts (a bit
like chromosomes). If two strains of flu infect the same cell, they can swap these
parts around. For example, if a human whos a pig farmer gets infected with a
swine flu virus at the same time as a regular human flu virus, the viruses that
come out of that person to infect another human might have a mix of some
human flu chromosomes and some swine flu chromosomes. This allows for a
much faster rate of change than if the virus was relying on regular mutations
alone.