24.05.15 DYSKINESIA OF GALL BLADDER D=1.559 E=1 24.05.15 RADICULITIS # F D=0.726 E=1 24.05.15 ALLERGY #J D=0.872 E=1 A disease group with an allergic affection of different sections of the respirat ory tract. Aetiology and pathogenesis result from allergic reactions both early and delayed. Both the respiratory tract as a whole and separate fragments can be affected, which, in turn, determines the allergosis form. 24.05.15 NODES TOXIC GOITER D=2.000 E=1 Hub toxic goiter - a disease characterized by the presence of node autonomously producing the elevated thyroid hormones, hyperplasia and reduced function of the remaining thyroid tissue. Usually occurs in women, usually aged over 40 years. In the pathogenesis of toxic goiter the primary role played by local disorder of the nervous impulses received by the individual sections of thyroid tissue. In contrast to diffuse toxic goiter in the pathogenesis of nodular goiter tiostimul iruyuschy long-acting factor (LATS) is not involved nodular goiter has often mal osimmpomnoe flow with moderately severe clinical picture (slight weakness, sligh t weight loss, moderate tachycardia) with no ophthalmopathy and myxedema pretibi alnoy. On palpation of the thyroid gland opredelyaktsya elastic unit with clear boundaries and a smooth surface, freely shifting swallowing. 24.05.15 DIFFUSE GOITRE # U D=0.897 E=1 Toxic diffuse goitre (Graves'-Basedow's disease) is a disease marked by the thyr oid gland's hyperplasia and hyperfunction. The disease may be prompted by heredi tary factors, infections, intoxications, psychemical traumas, etc. The foundatio n of pathogenesis is a disturbance of immune "supervision" resulting in the form ation of autoantibodies as a stimulating factor causing the gland's hyperfunctio n, hyperplasia and hypertrophy. Of significance here is changed sensitivity of tissues to thyroid hormones leading to a disturbance of metabolism between them. Clinical manifestations are dictated by a surplus of thyroid hormones affecting different kinds of metabolism, organs and tissues. Patients complain of irritab ility, weepiness, hypererethism, insomnia, flaccidity, fatigue, sweating, hands and body tremors. Patients lose weight despite good appetite (limosis). Young pa tients, on the contrary, may gain weight ("fat" Basedow). The thyroid gland is d iffusely enlarged, there being no relationship between its enlargement and serio usness of thyrotoxicosis. Changes in the eyes are as follows - bilateral exophth alm (no trophic disturbances), restricted movement of the eyeballs, Greffe sympt om (the upper eyelid lags behind the eyeball when looking down), Dalrimple sympt om (wide opening of the palpebral fissure), Mobius symptom (weakness of converge nce), and Kocher's symptom (retraction of the upper eyelid when shifting one's g aze). 2. Prescriptions: