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A r e a H e a lth
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H E A L T H
Intensive Care
Evidence Based Practice Guidelines
2004
Contraindications:
Severe aortic insufficiency as the balloon inflates, blood may be forced across the valve thereby overloading the ventricle and increasing cardiac work. Aortic aneurysm the increased pressure generated by counterpulsation may cause the aneurysm to rupture. Severe peripheral vascular disease may limit the ability to advance the catheter through atherosclerotic vessels. Severe coagulopathy
Complications:
Limb ischaemia due to occlusion of the femoral artery either by the catheter or by emboli from thrombus formation on the balloon Aortic dissection during insertion or rupture during pumping Haemorrhage from insertion site Helium emboli from the balloon Infection at site of insertion or catheter related
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Equipment: Insertion kit (introducer & sheath), intra-aortic balloon and console
ECG/pressure cable from external source into the IABP console Pressure monitoring set-up (transducer, pressure cable, saline, pressure bag) Major procedure tray, gowns, gloves, masks, goggles 10 ml syringe, 21 gauge needle, lignocaine 1% or 2% (without adrenaline) Sutures & sterile transparent dressing
Procedure:
Check the patients coagulation results Explain the procedure to the patient Wash hands, gather equipment, connect patient to monitor Baseline patient assessment: colour, temperature, capillary refill, pulses & sensation/movement of both legs Check that patient has patent IV access for administration of sedatives and emergency drugs Set-up pressure monitoring system to be connected to the central lumen of the catheter and the pump console. The arterial pressure waveform should appear on the monitor. Set initial timing for inflation & deflation C pressure artifact/positive overload B IAB inflation E IAB deflation A fill pressure baseline (10-15 MM Hg)
D plateau pressure
Low balloon pressure plateau could be caused by hypotension, hypovolaemia, low systemic vascular resistance, low balloon inflation volume, a balloon sized too small for the pt or positioned too low in the aorta.
Fig 3. Low balloon pressure plateau
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High balloon pressure plateau may be caused by hypertension, a balloon too large for the aorta, or a restriction to gas flow within the system. The top of the plateau may be squared or rounded.
Balloon pressure baseline elevation may be caused by a restriction of gas flow or gas system overpressurisation
Balloon pressure baseline depression usually indicates a helium leak. Other possible causes not related to helium leak include inappropriate timing settings (early inflation or late deflation) that do not permit enough time for gas to return to the console or a mechanical defect that causes failure to autofill.
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Timing continued: In early balloon inflation, the upstroke (arrow) of peak diastolic pressure occurs approx 9 little squares before the onset of systole.
In late balloon inflation a significant portion of the dicrotic notch (arrows) is visible.
In early deflation, note the U shape rather than V shape of the waveform and indication of a brief shelf (arrow) before the next systole.
In late deflation, the balloon remains partly or completely inflated at the beginning of the next systole. Note the balloon assisted aortic end-diastolic pressure (arrow) is greater than the unassisted pressure.
Late deflation is an extremely dangerous timing error because the LV must eject against the resistance imposed by the inflated balloon. Fig 11 Late balloon deflation Assisted aortic end-diastolic pressure
Peak
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Nursing Care:
Transduce the aortic arterial line (balloon lumen) to the Datascope pump and level with the patients mid-axillary line. For arterial flush bag use normal saline 500mls with heparin 1,000 u/s. Hourly monitoring of: heart rate and rhythm record systolic/diastolic/mean arterial pressures and diastolic augmentation (Use the BP obtained from the Datascope pump when titrating drugs) pedal pulses distal to the catheter site (Doppler may be necessary to assess pulse) L) radial pulse (If the catheter migrates forward it could compromise blood flow to the L) subclavian artery). colour, temperature and capillary refill sensation and movement of both lower extremities. Patient to be log rolled or jordan lifted and the end of the bed elevated no more than 300 to prevent catheter migration and arterial puncture. Careful monitoring of renal function (The catheter sits above the bifurcation of the renal arteries - backward migration may compromise blood flow to the kidneys). The balloon should not remain immobile for >20 minutes while insitu due to risk of thrombus formation. Assess insertion site each shift for redness, ooze Change dressing prn Carefully monitor the insertion site for signs of bleeding, infection, haematoma, or compartment syndrome of the affected limb. Heparinisation according to protocol may be initiated at 24 hours.
Weaning:
Counterpulsation may be reduced from 1:1 to 1:2 and finally 1:3 depending on the patients haemodynamics Do not set the pump at 1:3 unless for weaning and prior to removal. There is an increased risk of thrombus formation at counterpulsation of 1:3
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Removal:
Balloon catheters are removed by medical officers experienced in this procedure. When the catheter is removed, allow a very small volume of arterial bleed to occur to expel any small clots. Apply a pressure device eg Fem-stop to provide continuous pressure to the site for at least 30 minutes. Then cover site with a sandbag for 2-4 hours (with frequent checks for signs of bleeding). Contact Cardiology CNC in hours or A5a/b after hours for assistance if required.
Cardiac Arrest:
Switch to pressure triggering once pump alarms due to loss of ECG rhythm (remember to select "assist" after changing trigger modes). Reduce the pressure threshold if balloon fails to pump from pressure trigger (decrease arrows in auxiliary box under trigger options). The balloon pump does not need to be disconnected during defibrillation. If CPR cannot generate a consistent and reliable trigger, then switch to INTERNAL mode which will maintain movement of the IAB and therefore reduce the risk of thrombus formation.
WARNING: The use of INTERNAL trigger will produce asynchronous counterpulsation and should never be used in the event that the patient has an ECG or arterial pressure source available. Once the ECG or arterial signal has been reestablished, the trigger mode must be changed from INTERNAL to an acceptable patient trigger.
References: 1. Intra-aortic balloon pump learning package and resource file. Westmead Hospital. 2. The concepts of Intra-aortic balloon pumpimg. Datascope clinical support services, 1999. Datascope Medical Co. Ltd. 3. McVeigh, J.P., 1999. Principles and physiological effects of the intra-aortic balloon pump. NSW College of Nursing. 4. Staniatis, S.J., & Spandoni. S.M., 1997. Getting to the heart of IABP Therapy, RN.
Auburn Hospital and Community Health Services Blacktown Mt Druitt Health Service Cumberland Hospital Lottie Stewart Hospital St Josephs Hospital Westmead Hospital and Community health Services Parramatta Linen Services