PROVIDING NEW POSSIBILITIES IN THE QUEST FOR WELLNESS

Dr.Praveen Kumaar Saxenaa

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Todays number one health hazard is arteriosclerosis or hardening of our arteries. This causes heart attacks, strokes, and peripheral vascular disease (usually in the legs) which leads to more misery and suffering than any other disease known today.

MBBS,DMRD(OSM) FACAM ,FRCAM CLINICAL METAL TOXICOLOGIST (IBCMT)

Our great great grand parents and their parents had very little arteriosclerosis even though their diets included foods known to be high in cholesterol such as eggs, butter, lard, etc. !owever,they did not eat any hydrogenated oils, and their grain foods were home ground and not processed. what is affecting you inspite of maintaining a normal cholesterol in blood !eavy metals like cadmium, aluminum, lead, mercury and nickel and more are so pervasive in our society today. These metals are added to our food chain from many industrial sources including pesticides, everyday products, amalgam "silver" dental fillings, the air we breathe and the water we drink. They are virtually everywhere, slowly absorbing and building up inside our bodies and eventually causing disease. The body becomes overwhelmed with so many to#ins that it can no longer completely metabolize them. !eavy metal to#ins such as lead, mercury, aluminum, cadmium, arsenic etc. has been clinically proven to damage and cause endothelial dysfunction. $umerous established studies have confirmed that an impaired endothelial function is linked to all ma%or coronary heart diseases. &therosclerosis is the main cause for heart attack and strokes. The endothelium is inner lining of blood vessels. This lining tissue generates the powerful arterial vessel dilator nitric o#ide ($.O.).
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PROVIDING NEW POSSIBILITIES IN THE QUEST FOR WELLNESS

Dr.Praveen Kumaar Saxenaa

MBBS,DMRD(OSM) FACAM ,FRCAM CLINICAL METAL TOXICOLOGIST (IBCMT)

The endothelium also produces prostacyclin which slows the clotting of blood and also causes beneficial dilating of arteries. The third important endothelial product is heparin which prevents clots from forming without causing bleeding. '#cessive deposition of heavy metals in the endothelium diminishes the endothelium(s ability to produce nitric o#ide, prostacyclin, and heparin. Therefore, heavy metal to#icity leads to decreased amounts of $itric O#ide, which leads to unrela#ed blood vessels and associated decreased blood flow, &) *'++ as vulnerability to infection of fresh cholesterol by low grade virus, such as !erpes, that can form and break a vesicle within an artery and cause an immediate !ypercoagulable state, with a subse,uent blood clot formation and sudden death. 'ndothelial cells play a vital role in the health and integrity of every tissue of the body. $.O. is a potent vasodilator and a strong anti o#idant. *hen the endothelium is damaged, $.O. production is reduced. This leads to the reduction of vasodilatation, or conversely, an increase in vascular constriction. -educed $.O. production, as a result of to#ic metal insult, leads to a reduction in vascular lumen size, restriction of blood flow, and ultimately an increase in blood pressure. This means, in layman(s terms, an increased risk of stroke and heart attack. ./ yrs male patient c0o angina and essential hypertension. long history of essential hypertension. !e was told at about age /1 that he had "high blood pressure" with a systolic blood pressure of /12 mm0!g. The history also revealed that both his mother and father died at age 34 of myocardial infarcton. +ater, on a physical e#amination with a different physician, his blood pressure was a /520/22 mm0!g and he was diagnosed with essential hypertension. !is medication consisted of daily doses of beta blockers 6 calcium channel blockers . !e continued on medication over a period of fifteen years with fluctuations in his blood pressure.
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PROVIDING NEW POSSIBILITIES IN THE QUEST FOR WELLNESS

Dr.Praveen Kumaar Saxenaa

MBBS,DMRD(OSM) FACAM ,FRCAM CLINICAL METAL TOXICOLOGIST (IBCMT)

*hen first seen at the 7entre, his blood pressure was /8201. with medication9 /120/22 without medication. !e was given an initial intravenous chelation :re and post chelation ;8 hour urine samples were collected and aluminum, cadmium, lead and mercury levels measured. The lead level was of particular interest as several published studies demonstrated a relationship of lead levels and hypertension. !is pre chelation urine lead level was /8.2 mg0;8 hours. The post chelation urine lead level was 4/ mg0;8 hours. The 7entre considers a . fold increase in urine lead e#cretion after chelation an indication of increased body load of lead. !e was then started on a series of intravenous infusions administered at appro#imately weekly intervals over a period of seven months. 'ach treatment was transfused over a period of < . hours. Other post chelation urine studies were performed over a period of time and showed urine lead level of <4, 82, and .2 mg0;8 hours respectively. 7omplete blood counts, urinalysis and chemistry profile were done throughout the treatment and showed no adverse effects of the treatment. The patient slowly decreased his medication during the treatment and stopped them completely at the last chelation (=arch /8,;2/2). !is blood pressure at the last chelation was /;8018 mm0!g. !e has not taken any blood pressure medication since the last chelation treatment.

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