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Pulse: Rapid and weak BP: Hypotensive Cardiac output is decreased therefore the following clinical manifestations occur: Angina Syncope Generalized weakness Dizziness Shortness of breath Changes in mental status, starting with confusion and restlessness, leading to unconsciousness. Conscious sensation of ineffective cardiac activity often accompanied by anxiety. Presence and severity of the signs and symptoms depends on rhythm duration.
Initial treatment is based on the presence or absence of a palpable pulse. If hemodynamically stable, with a pulse, drug intervention is appropriate e.g., procainamide, amiodarone, lidocaine. If hemodynamically unstable (pulseless) defibrillation and cardiopulmonary resuscitation (CPR) is indicated. After initial stabilization other treatments which may be considered include: Implantable cardioverter defibrillator Electrophysiology Studies (EPS): ventricles are stimulated to produce VT: then antiarrhythmic drugs are administered, followed by a second attempt to produce the VT. If unable to reproduce the dysrhythmia, the drug is considered effective and continuous therapy is instituted. Drugs used include: procainamide, amiodarone, and lidocaiane. Radio-frequency ablation, burning the area or focus in the ventricle where the VT is coming from may also be considered. Chapter 39 has a complete description of EPS and ablation.
CHART 3810
Dysrhythmia Torsade de pointes (TdP)
Severe myocardial ischemia Coronary heart disease Myocardial infarction Advanced heart block Abnormal repolarization Vagal stimulation Drug toxicity, e.g., psychotropics, digoxin Metabolic abnormalities, e.g., hypokalemia, hypomagnesemia Hypoxia Trauma Terminal event in many disease states Electrical shock Profound hypovolemia Massive myocardial damage Excessive vagal tone due to loss of sympathetic tone Obstruction of blood flow to or from the heart, e.g., severe pulmonary embolism Pericardial tamponade Myocardial rupture Massive cardiac trauma resulting in cardiac tamponade and/or tension pneumothorax Severe acidosis Hyperkalemia Hypothermia Drug overdose, e.g., tricyclics, betablockers, calcium channel blockers, digoxin Cardiac standstill from massive cardiac muscle damage
Pulse: None palpable BP: None Unconscious Seizures may occur Death, if untreated
Assess for pulse and blood pressure; initiate CPR protocol per ACLS guidelines. Find cause and treat.
Asystole
Check the rhythm in two leads in order to rule out the possibility of fine ventricular fibrillation. Also, check the lead placement to make sure it has not fallen off. Initiate CPR and ACLS protocols. Administer drugs per ACLS guidelines, agency protocol or health care providers orders.