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Question
Is the following statement true or false? Blood pressure in arteries is higher than blood pressure in veins.
Answer
True Rationale: Venous return to the heart exists in a low-pressure system in which backflow is prevented by valves. Arteries and arterioles are higher-pressure blood vessels.
Anatomy and Physiology of the Heart Coronary Arteries The Cardiac Conduction System
Question
The student nurse is assessing a patient who has a dysrhythmia. The student understands that electrical conduction of the heart usually originates in the SA node. What sequence completes the conduction? SA node to bundle of HIS to AV node to Purkinje fibers SA node to AV node to Purkinje fibers to bundle of HIS SA node to bundle of His to Purkinje fibers to AV node SA node to AV node to bundle of His to Purkinje fibers
Answer
D. SA node to AV node to bundle of His to Purkinje fibers Rationale: The normal cardiac conduction route proceeds from the SA node to the AV node. It then goes from the bundle of His to the Purkinje fibers.
Cardiac Output
CO = stroke volume (SV) heart rate (HR) SV is influenced by three interdependent factors: Preload: The pressure generated in the ventricles at the end of diastole and the resultant stretching of the muscle fibers Afterload: The amount of resistance to ejection of blood from the ventricle Contractility: The force generated by the contracting myocardium under any given condition
Assessment Parameters
Health history Family history Medications Nutrition and metabolism Elimination Activity
Physical Assessment
Integumentary inspection and palpation Blood pressure: Pulse pressure Orthostatic changes Arterial pulses: Rate, rhythm, and quality Inspection of jugular venous pulsations Assessment of lungs and abdomen
Question
A patient's systolic pressure is 122 mm Hg and diastolic pressure is 75 mm Hg. What would the patients pulse pressure be? 122 98 197 47
Answer
D. 47 Rationale: Pulse pressure is the difference between the systolic and the diastolic pressures.
Locating and palpating apical pulse Heart Sound Physiology Heart Gallop Sound Diagnostic Evaluations
Cardiac biomarkers: Creatine kinase (CK) / CK-MB Myoglobin Troponin T and I LDL HDL Triglycerides Brain (B-type) natriuretic peptide (BNP)
Central venous pressure monitoring Pulmonary artery pressure monitoring Intra-arterial blood pressure monitoring Doppler ultrasound studies
Pathophysiology of Hypertension
BP is the product of cardiac output multiplied by peripheral resistance Peripheral vascular resistance (PVR) is related to the diameter of the blood vessel and the viscosity of the blood Management aims to decrease peripheral resistance, blood volume, or the strength, force, and rate of myocardial contraction 95% of patients have primary hypertension
D. Dyslipidemia Rationale: Hypertension often accompanies other risk factors for atherosclerotic heart disease, such as obesity, diabetes mellitus, metabolic syndrome, and a sedentary lifestyle. Electrolyte imbalances and dysrhythmias are not identified as risk factors for hypertension.
Complications of Hypertension
Blood vessel damage (heart, kidneys, brain, and eyes) Myocardial infarction Heart failure Left ventricular hypertrophy Renal failure Stroke
Impaired vision
Hypertensive Crises
Defined as a systolic blood pressure of greater than 180 mm Hg or a diastolic blood pressure of greater than 120 mm Hg Hypertensive emergency is a situation in which BP is higher than 180/120 mm Hg and must be lowered quickly to halt or prevent damage to the target organs Hypertensive urgency describes a situation in which BP is severely elevated but there is no evidence of impending or progressive target organ damage Both are managed with continuous intravenous infusion of a short-acting titratable antihypertensive agent
Chapter 14 Nursing Management: Patients With Coronary Vascular Disorders Coronary Atherosclerosis
The most common cause of cardiovascular disease Atheromas or plaques protrude into the lumen of the vessel Thrombi may form and obstruct blood flow, leading to sudden cardiac death or an acute MI The anatomic structure of the coronary arteries makes them particularly susceptible to the mechanisms of atherosclerosis
Angina Pectoris
Almost always associated with a significant obstruction of a major coronary artery Anginal pain can have widely varying characteristics Diagnosis is usually by history, ECG, and cardiac biomarker analysis
Question When discussing angina pectoris secondary to atherosclerotic disease with a patient, the patient asks why she experiences chest pain with exertion. The nurse informs the patient that exertion: Increases the heart's oxygen demands Causes vasoconstriction of the heart Increases blood flow to the mesenteric area Reduces the effectiveness of medications Answer A. Increases the heart's oxygen demands Rationale: Physical exertion increases the myocardial oxygen demand. If the patient has arteriosclerosis of the coronary arteries, then blood supply is diminished to the myocardium, resulting in pain. Exercise does not cause vasoconstriction, reduced effectiveness of medications, or increased blood flow to the mesenteric area.
Qualification criteria
Bypass graft veins CABG Cardiopulmonary Bypass Nursing Process: The Postoperative Cardiac Surgery Patient
Initial postoperative care focuses on hemodynamic stability and recovery from general anesthesia Later care focuses on the monitoring of cardiopulmonary status, pain management, wound care, progressive activity, and nutrition Frequent, multisystemic assessment is imperative
Chapter 15
Nursing Management: Patients With Complications From Heart Disease
Types of HF
Systolic heart failure: Characterized by a weakened heart muscle. Diastolic heart failure: Characterized by a stiff and noncompliant heart muscle Assessment of the ejection fraction (% of blood ejected with each contraction) is performed to assist in determining the type of HF Question Is the following statement true or false? A patient with systolic HF is likely to have a heart that is unable to contract strongly enough to ensure adequate circulation and tissue perfusion. Rationale: In systolic HF, the patients heart muscle is too weak to meet the patients complete metabolic needs.
Pathophysiology
HF results from various underlying factors and the compensatory mechanisms that are put in place Compensatory mechanisms tend to ultimately exacerbate the signs and symptoms of HF
Risk Factors
Major risk factors: Age, male sex, hypertension, left ventricular hypertrophy, myocardial infarction, valvular heart disease, and obesity Minor risk factors: Excessive alcohol consumption, smoking, high cholesterol, diabetes, toxins, sleep-disordered breathing, chronic kidney disease, low socioeconomic status, psychological stress, sedentary lifestyle, and genetics
Question
The nurses assessment of an older adult patient reveals multiple risk factors for HF. Which of the following risk factors should the nurse address in patient teaching? The patients age The patients racial background The patients sex The patients diabetes management
Answer
D. The patients diabetes management Rationale: Age, sex, and race are nonmodifiable risk factors for HF. Diabetes management is an area that is amenable to education and nursing interventions.
Management of HF
Lifestyle changes Medications: ACE inhibitors, beta blockers, diuretics, and digitalis Nursing assessment prioritizes symptoms of pulmonary and systemic fluid overload, health history, and monitoring of intake and output Question You are writing a teaching plan for a patient diagnosed with heart failure. What would be a priority inclusion in the teaching plan? Self-care Cardiac rehabilitation Dressing changes Nutrition Answer A. Self-care Rationale: Long-term management of HF requires extensive self-care by patients; nutrition would be one aspect of this self-care. Cardiac rehabilitation is more commonly used in the recovery of patients who have undergone cardiac surgery. Dressing changes are not relevant to HF.
Pulmonary Edema
Pathophysiology Clinical manifestations and assessment Management of symptoms follows the similar clinical management plan for treating acute decompensated heart failure
Cardiogenic Shock
Occurs when decreased CO leads to inadequate tissue perfusion and initiation of the shock syndrome Pathophysiology Manifestations Management