Diabetes is primarily a disorder of Google's metabolism related to absent when suction insulin supply or poor utilization of the insulin

that is available to most common types of diabetes are type I and type II insulin needs a hormone produced by the beta cells in the islets of Langerhans of the pancreas in Salinas continue series and the gutsy stream in small pulsatile increments hormones that were that work to oppose the effects of insulin are glucagon phenanthrene growth hormone and cortisol in a cold country of regular regulatory hormones, These hormones work to increase blood glucose levels by stimulating glucose production output by the liver and by decreasing the movement of glucose into the cells easily did is read released from the pancreatic beta cells as it is a precursor, pro-insulin and then is routed to the liver it promotes glucose transport from the bloodstream across the cell membrane to the cytoplasm of the cell falling insulin level during the old normal overnight fasting facilitates the release of stored glucose from the liver protein from the muscle and fat from the adipose tissue squiggle muscle and adipose tissue specific receptors for insulin and are considered insulin-dependent tissue other tissue example brain liver blood cells: dedicated in an incidental glucose transport required Dick would glucose apply for normal function Type I diabetes mellitus storm really known as juvenile onset or insulin-dependent diabetes it results from the progressive destruction of the pancreatic beta cells due to an autoimmune process in susceptible individuals type I may be caused by non-immune factors of unknown etiology is known as type IP diabetes. When type I diabetes is caused by an immune mechanism, the disease is known as type IA predisposition to type I diabetes is believed related to human leukocyte antigens [HLA]. An individual with Satan each LE types is exposed to viral infections, the beta cells of the pancreas are destroyed type I is associated the long preclinical. The islets cell autoantibodies responsible for beta cell destruction a present from months to years before the onset of symptoms. The client has a history of recent and sudden weight loss as well as the classics and does polydipsia bracket excessive thirst bracket close, polyuria bracket open frequent urination got close, and polling fascia bracket open access of hunger bracket close. Type I patients require supply when Celine from an outside source such as an section, in order to sustain life without incident, the client will develop that Betty P2 acidosis a life-threatening condition resulting in metabolic acidosis that is untreated could be thoughtful eventually is more beta cells and destroyed, Doug glucose levels increase, more incidents needed, honey. Over Prediabetes it's also known as impaired glucose tolerance or impaired fasting glucose when a fasting order to our plasma glucose level is higher than normal but lower than that considered diagnostic for diabetes this puts people at risk of developing diabetes within 3 to 5 years long-term damage to the body specially the heart and blood vessels may already be occurring clients prediabetes Type II Diabetes Mellitus usually occurs over 35 years of each MDT to 90% of clients are over. At the time of diagnosis it has attendance sheet to run in families him him him. In type II diabetes the pantry is usually continued to produce some endogenous self-made incident however, it the insulin is insufficient for the needs of the body, is poorly utilized by the tissue, or both in contrast, there is a virtual absence of endogenous insulin in type I diabetes four major metabolic abnormalities that have a role in the development of type II diabetes 1. In him the first factor is insulin resistance, which is a condition of the body to respond to the action of insulin it is you insulin receptors that are unresponsive to the action of insulin, inception and number, or both this results in the entry of glucose into the cells being impeded, resulting hyperglycemia. 2. Second factor in the development of type II diabetes is a marked decrease in the ability of the pancreas produce insulin, as a beta cells become fatigued from the Compton sent safely or production of incident 3. The third factor is inappropriate glucose production by the liver which it does it in a haphazard way that does not correspond to the body's needs at the time.

4. The fourth is the alteration in the production of hormones and cytokines by adipose tissue and people kinds of peer-to-peer Wynkoop wasn't Metabolism I like the country to the pathophysiology of type II diabetes metabolic syndrome is a cluster of abnormalities that act synergistically to greatly increase risk of cardiovascular disease. Metabolic syndrome is characteristic characterized by abnormal obesity, hypertension, dyslipidemia, insulin resistance, and displacing Gestational diabetes GDM develops during pregnancy detected between 24 to 28 weeks of dissertation following an oral glucose tolerance test treatment of gestational diabetes reduces perinatal death in neonatal complications nutritional counseling is the first line of therapy physical activity should be encouraged Secondary diabetes this is diabetes occurring to them that due to another medical condition or treatment a result of the treatment of a medical condition that causes abnormal glucose levels. These conditions include schizophrenia, cystic fibrosis, hyperthyroidism, immunosuppressive therapy, and the use of parenteral nutrition.