y Diminished or absent insulin production. y Most often at age of 15 y.o.

y Autoimmune destruction of pancreatic islets y Familial and lifelong

Type I/ IDDM

Classification

Type II/NIDDM

y Hormone produce by the placenta has insulin resistance y 4% of pregnant women y Goes away during delivery y May develop Type II after 2 years

Gestational

y Insufficient insulin production y Ketoacidosis not common y Adult and 40 y.o. mostly y Familial y Obesity creates insulin demand that cannot be met by amount of circulating insulin present y May need insulin

Diabetes Mellitus
Assessment
Complica tions
T R E A T M E N T

3Ps
Type I & II

Type I &II

Insulin
y Hypoglycemia y Lipodystrophy y Hormone counter Action

Poorly Controlled Diabetes y Diabetic Ketoacidosis y Hyperosmolar Hyperglycemia (now Ketotic Coma) y Electrolyte Imbalance

Type I: weight thirst Bed wetting Rapid onset

Type II: weight Eye problem Slow onset

y Polyphagia (much eating) y Polydipsia ( thirst) y Polyuria ( urine volume)

Long Term
Angiopathy Peripheral Vascular Disease Retinopathy Nephropathy Neuropathy Infection insulin need, Glucose storage, Glucose Fluctuation

Dx

y Insulin y Oral Hypoglycemic y Diet y Exercise

y Fasting (above 125mg/100cc) y Glucose Tolerance Test ( 2 hour value greater than 200 mg/dl) y Glycosylated Hemoglobin is

Submitted by: Leslie Marie D. Rendon BSN II-Beneficence