CARBOHYDRATES (Seminar |vitn} 4 (print name) points) Y 1 Which ofthe following glucose transporter: iif ) pee in GLUT 1 \ enakh deskine BY GLUT 3 as D. GLUT 4 ssulw cops dm E GLUTS ssupllateret Intestinal absorption of fructose into the mucosal els is de to: ee i ‘Na ~dependent secondary active at C. insulin-dependent facilitated diffusion 1D. primary active transport depend upon hydrolisis orate E, Ca¥ *-dependent secondary active transport 3. Se Se ey é saliva /) 3B: gastric secretions c lumen of the small intestinevind) Am in the diagram corresponds 2. Which part of the cartilage proteoglycan shown in the diagram to hyaluronic ue acid: S IaOuinked lysapenteinsthe gr residue tached theaming aid sua ‘yiglucosamine 5 C. N-acetyIneuraminic acid x D. glucose E. galactose 9. How would affected by(ibition of alyransterass) “A. dolichol pl }osphate recycling would be blocked B. the pentasaccharide core would not be formed 4 C. serine or threonine hydroxyl groups would not be glycosylated D. monosaccharides would not be linked to the amide-N’ of as; i "© ea ser eae SEU A. the synthesis of polysai iccharides with altered structures that results in abnormal cell shape and functioni B. the synthesis of glycosaminoglycans with ‘abnormal structures that results in ab /\ cell shape and functioning clevated biosynthesis and accumulation of glycosaminoglycans efective enzymes involved in the . ‘synt reduced levels of these ‘molecules\ “san Carbohydrates (Seminar IX) Bo Jucurte) grou) (pots) b E-a-amylase wv E. increase blood presse 3. Several hours afier a meal, when blood glucose levels are vines Vi, cmA. glucose 6-phosphatase. * ~ B. glucose phosphate isomerase. C. phoshofructokinase-1.